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Researcher · University of Siena · Italy

Emanuele Gabellieri

Emanuele Gabellieri publishes mostly in Molecular Biology, Physiology and Pharmacology, on topics such as Alzheimer's disease research and treatments, Computational Drug Discovery Methods and Cholinesterase and Neurodegenerative Diseases.

ORCID 0000-0003-2241-8611 OpenAlex
World rank
#3,214,225
of 7,710,434 ranked researchers
Rank in Italy
#108,789
of 242,206
Works
16
Citations
704
Citations per work
44.0
h-index
12
i10-index 12Top 65% in Molecular Biology
Emanuele Gabellieri

What does Emanuele Gabellieri research?

Emanuele Gabellieri's work is mostly in Medicine (29%), Biochemistry, Genetics and Molecular Biology (19%), Chemistry (12%).

Health Sciences: 5 works · 29.2% of their workMedicine: 5 works · 29.2% of their workPharmacology: 2 works · 14.6% of their workPhysiology: 2 works · 14.6% of their workLife Sciences: 4 works · 25.1% of their workBiochemistry, Genetics and Molecular Biology: 3 works · 18.8% of their workMolecular Biology: 3 works · 18.8% of their workNeuroscience: 1 works · 6.3% of their workCellular and Molecular Neuroscience: 1 works · 6.3% of their workPhysical Sciences: 4 works · 25.0% of their workChemistry: 2 works · 12.5% of their workOrganic Chemistry: 2 works · 12.5% of their workComputer Science: 2 works · 12.5% of their workComputational Theory and Mathematics: 2 works · 12.5% of their workEverything else: 3 works · 20.7% of their workTHEIR WORKEmanueleGabellieri16works
Life Sciences25%Physical Sciences25%Health Sciences29%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Medicine529.2%
    1. Biochemistry, Genetics and Molecular Biology318.8%
    2. Neuroscience16.3%
    1. Chemistry212.5%
    2. Computer Science212.5%
  4. Everything else320.7%
  1. 1Molecular Biology19% of their works
  2. 2Physiology15% of their works
  3. 3Pharmacology15% of their works
  4. 4Computational Theory and Mathematics12% of their works
  5. 5Organic Chemistry12% of their works
  6. 6Cellular and Molecular Neuroscience6% of their works

Research topics

Which keywords describe Emanuele Gabellieri's research?

The keywords of their largest research topics: Oxidative Stress, Neurodegenerative Diseases, Pharmacology, Amyloid Hypothesis, Tau Pathology, Acetylcholinesterase, Cholinesterase Inhibitors and Molecular Docking.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most.

All 20 words, with their numbers
  1. Oxidative Stress92 topics
  2. Neurodegenerative Diseases82 topics
  3. Pharmacology82 topics
  4. Amyloid Hypothesis71 topic
  5. Tau Pathology71 topic
  6. Acetylcholinesterase61 topic
  7. Cholinesterase Inhibitors61 topic
  8. Molecular Docking61 topic
  9. Virtual Screening61 topic
  10. Drug Discovery52 topics
  11. Foldamers31 topic
  12. Neurotransmission31 topic
  13. Peptide Synthesis31 topic
  14. Synaptic Plasticity31 topic
  15. Cerebral Hypoperfusion21 topic
  16. Click Chemistry21 topic
  17. Copper-Catalyzed Cycloaddition21 topic
  18. Marine Natural Products21 topic
  19. Sponge-Associated Microorganisms21 topic
  20. Vascular Cognitive Impairment21 topic

Where does Emanuele Gabellieri work?

Emanuele Gabellieri's main affiliation in the publication record is University of Siena, Italy.

How many publications and citations does Emanuele Gabellieri have?

OpenAlex counts 16 works and 704 citations for Emanuele Gabellieri, an h-index of 12, ranking #3,214,225 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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