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Researcher · Universidade do Extremo Sul Catarinense · Brazil

Felipe Dal‐Pizzol

Felipe Dal‐Pizzol publishes mostly in Critical Care and Intensive Care Medicine, Molecular Biology and Epidemiology, on topics such as Intensive Care Unit Cognitive Disorders, Neuroinflammation and Neurodegeneration Mechanisms and Sepsis Diagnosis and Treatment.

ORCID 0000-0003-3003-8977 OpenAlex
World rank
#378,681
of 7,710,434 ranked researchers
Rank in Brazil
#1,680
of 253,246
Works
631
Citations
23k
Citations per work
36.1
h-index
82
i10-index 382Top 0.5% in Critical Care and Intensive Care Medicine
Felipe Dal‐Pizzol

What does Felipe Dal‐Pizzol research?

Felipe Dal‐Pizzol's work is mostly in Medicine (14%), Neuroscience (9%), Biochemistry, Genetics and Molecular Biology (7%).

Everything else: 406 works · 64.4% of their workLife Sciences: 137 works · 21.7% of their workNeuroscience: 58 works · 9.2% of their workNeurology: 34 works · 5.4% of their workCellular and Molecular Neuroscience: 24 works · 3.8% of their workBiochemistry, Genetics and Molecular Biology: 45 works · 7.1% of their workMolecular Biology: 45 works · 7.1% of their workImmunology and Microbiology: 34 works · 5.4% of their workImmunology: 34 works · 5.4% of their workHealth Sciences: 88 works · 13.9% of their workMedicine: 88 works · 13.9% of their workCritical Care and Intensive Care Medicine: 45 works · 7.2% of their workEpidemiology: 42 works · 6.7% of their workTHEIR WORKFelipe Dal‐Pizzol631works
Life Sciences22%Health Sciences14%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
  1. Everything else40664.4%
    1. Neuroscience589.2%
    2. Biochemistry, Genetics and Molecular Biology457.1%
    3. Immunology and Microbiology345.4%
    1. Medicine8813.9%
  1. 1Critical Care and Intensive Care Medicine7% of their works
  2. 2Molecular Biology7% of their works
  3. 3Epidemiology7% of their works
  4. 4Immunology5% of their works
  5. 5Neurology5% of their works
  6. 6Cellular and Molecular Neuroscience4% of their works

Research topics

Which keywords describe Felipe Dal‐Pizzol's research?

The keywords of their largest research topics: Microglia, Neuroinflammation, Delirium, Intensive Care Unit, Epidemiology, Depression, Astrocytes and Neurodegeneration.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 20 words, with their numbers
  1. Microglia1322 topics
  2. Neuroinflammation1322 topics
  3. Delirium1021 topic
  4. Intensive Care Unit1021 topic
  5. Epidemiology962 topics
  6. Depression902 topics
  7. Astrocytes771 topic
  8. Neurodegeneration771 topic
  9. Sepsis711 topic
  10. Septic Shock711 topic
  11. Immune Activation551 topic
  12. Tryptophan Metabolism551 topic
  13. Pattern Recognition Receptors501 topic
  14. Toll-like Receptors501 topic
  15. Brain351 topic
  16. Stress351 topic
  17. Neurotransmission321 topic
  18. Synaptic Plasticity321 topic
  19. Clinical Characteristics251 topic
  20. SARS-CoV-2251 topic

Where does Felipe Dal‐Pizzol work?

Felipe Dal‐Pizzol's main affiliation in the publication record is Universidade do Extremo Sul Catarinense, Brazil.

How many publications and citations does Felipe Dal‐Pizzol have?

OpenAlex counts 631 works and 22,775 citations for Felipe Dal‐Pizzol, an h-index of 82, ranking #378,681 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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