Fernanda G. De Felice
Fernanda G. De Felice publishes mostly in Physiology, Molecular Biology and Cellular and Molecular Neuroscience, on topics such as Alzheimer's disease research and treatments, Neuroinflammation and Neurodegeneration Mechanisms and Neuroscience and Neuropharmacology Research.
- World rank
- #28,032 of 7,710,434 ranked researchers
- Rank in Brazil
- #62 of 253,246
- Works
- 168
- Citations
- 17k
- Citations per work
- 103.1
What does Fernanda G. De Felice research?
Fernanda G. De Felice's work is mostly in Medicine (29%), Neuroscience (18%), Biochemistry, Genetics and Molecular Biology (14%).
Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.
The fields and subfields, with their numbers
- Everything else6639.0%
- Life Sciences5432.0%
- Neuroscience3018.0%
- Biochemistry, Genetics and Molecular Biology2414.0%
- Health Sciences4929.0%
- Medicine4929.0%
- 1Physiology22% of their works
- 2Molecular Biology14% of their works
- 3Cellular and Molecular Neuroscience9% of their works
- 4Neurology9% of their works
- 5Pharmacology4% of their works
- 6Neurology3% of their works
Research topics
- Alzheimer's disease research and treatments
- Neuroinflammation and Neurodegeneration Mechanisms
- Neuroscience and Neuropharmacology Research
- Adipose Tissue and Metabolism
- Cholinesterase and Neurodegenerative Diseases
- Tryptophan and brain disorders
- Computational Drug Discovery Methods
- Mitochondrial Function and Pathology
Which keywords describe Fernanda G. De Felice's research?
The keywords of their largest research topics: Neuroinflammation, Neurodegeneration, Oxidative Stress, Amyloid Hypothesis, Tau Pathology, Cytokines, Microglia and Astrocytes.
- Reactive Oxygen Species
- Virtual Screening
- Tryptophan Metabolism
- Cholinesterase Inhibitors
- Thermogenesis
- Synaptic Plasticity
- Neurodegenerative Diseases
- Metabolic Regulation
- Astrocytes
- Cytokines
- Amyloid Hypothesis
- Neurodegeneration
- Neuroinflammation
- Oxidative Stress
- Tau Pathology
- Microglia
- Synaptic Pruning
- Mitochondrial Dysfunction
- Neurotransmission
- Brown Adipose Tissue
- Acetylcholinesterase
- Depression
- Molecular Docking
- Mitochondria
Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.
All 24 words, with their numbers
- Neuroinflammation1243 topics
- Neurodegeneration1102 topics
- Oxidative Stress1023 topics
- Amyloid Hypothesis761 topic
- Tau Pathology761 topic
- Cytokines482 topics
- Microglia482 topics
- Astrocytes341 topic
- Synaptic Pruning341 topic
- Metabolic Regulation302 topics
- Mitochondrial Dysfunction302 topics
- Neurodegenerative Diseases292 topics
- Neurotransmission271 topic
- Synaptic Plasticity271 topic
- Brown Adipose Tissue181 topic
- Thermogenesis181 topic
- Acetylcholinesterase171 topic
- Cholinesterase Inhibitors171 topic
- Depression141 topic
- Tryptophan Metabolism141 topic
- Molecular Docking131 topic
- Virtual Screening131 topic
- Mitochondria121 topic
- Reactive Oxygen Species121 topic
Where does Fernanda G. De Felice work?
Fernanda G. De Felice's main affiliation in the publication record is Universidade Federal do Rio de Janeiro, Brazil.
How many publications and citations does Fernanda G. De Felice have?
OpenAlex counts 168 works and 17,326 citations for Fernanda G. De Felice, an h-index of 66, ranking #28,032 of 7,710,434 researchers worldwide on the quality score.
Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.
Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.
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