Günter U. Höglinger
Günter U. Höglinger publishes mostly in Neurology, Physiology and Cellular and Molecular Neuroscience, on topics such as Parkinson's Disease Mechanisms and Treatments, Neurological disorders and treatments and Alzheimer's disease research and treatments.
- World rank
- #53,506 of 7,710,434 ranked researchers
- Rank in Germany
- #3,048 of 364,343
- Works
- 603
- Citations
- 33k
- Citations per work
- 54.9
What does Günter U. Höglinger research?
Günter U. Höglinger's work is mostly in Medicine (49%), Neuroscience (15%), Biochemistry, Genetics and Molecular Biology (6%).
Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.
The fields and subfields, with their numbers
- Health Sciences29348.6%
- Medicine29348.6%
- Everything else17929.7%
- Life Sciences13121.7%
- 1Neurology34% of their works
- 2Physiology10% of their works
- 3Cellular and Molecular Neuroscience9% of their works
- 4Molecular Biology6% of their works
- 5Neurology6% of their works
- 6Radiology, Nuclear Medicine and Imaging4% of their works
Research topics
- Parkinson's Disease Mechanisms and Treatments
- Neurological disorders and treatments
- Alzheimer's disease research and treatments
- Genetic Neurodegenerative Diseases
- Neurological diseases and metabolism
- Dementia and Cognitive Impairment Research
- Amyotrophic Lateral Sclerosis Research
- Nuclear Receptors and Signaling
Which keywords describe Günter U. Höglinger's research?
The keywords of their largest research topics: Neurodegeneration, Parkinson's Disease, Oxidative Stress, Mitochondrial Dysfunction, Dopaminergic Neurons, a-Synuclein, Genetic Risk Factors and Deep Brain Stimulation.
- Nurr1
- TDP-43
- Mild Cognitive Impairment
- Pantothenate Kinase
- Protein Aggregation
- Neuroimaging
- Amyloid Hypothesis
- Deep Brain Stimulation
- Genetic Risk Factors
- Mitochondrial Dysfunction
- Parkinson's Disease
- Neurodegeneration
- Oxidative Stress
- Dopaminergic Neurons
- a-Synuclein
- Subthalamic Nucleus
- Tau Pathology
- Huntington's Disease
- Brain Iron Accumulation
- Alzheimer's Disease
- FUS/TLS
- NR4A2
Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.
All 22 words, with their numbers
- Neurodegeneration6015 topics
- Parkinson's Disease5083 topics
- Oxidative Stress4682 topics
- Mitochondrial Dysfunction4533 topics
- Dopaminergic Neurons3672 topics
- Genetic Risk Factors3321 topic
- a-Synuclein3321 topic
- Deep Brain Stimulation1411 topic
- Subthalamic Nucleus1411 topic
- Amyloid Hypothesis1361 topic
- Tau Pathology1361 topic
- Neuroimaging1052 topics
- Huntington's Disease631 topic
- Protein Aggregation631 topic
- Brain Iron Accumulation581 topic
- Pantothenate Kinase581 topic
- Alzheimer's Disease471 topic
- Mild Cognitive Impairment471 topic
- FUS/TLS401 topic
- TDP-43401 topic
- NR4A2351 topic
- Nurr1351 topic
Where does Günter U. Höglinger work?
Günter U. Höglinger's main affiliation in the publication record is German Center for Neurodegenerative Diseases, Germany.
How many publications and citations does Günter U. Höglinger have?
OpenAlex counts 603 works and 33,076 citations for Günter U. Höglinger, an h-index of 85, ranking #53,506 of 7,710,434 researchers worldwide on the quality score.
Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.
Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.
Discussion
Ask a question or share a note about Günter U. Höglinger. Keep it to the research: no personal attacks, advertising or links. Any reader can report a post, which collapses it for everyone until it is opened.