Halil Önder
Halil Önder publishes mostly in Neurology, Cellular and Molecular Neuroscience and Psychiatry and Mental health, on topics such as Neurological disorders and treatments, Parkinson's Disease Mechanisms and Treatments and Cerebrospinal fluid and hydrocephalus.
- World rank
- #7,706,225 of 7,710,434 ranked researchers
- Rank in Türkiye
- #117,567 of 117,622
- Works
- 292
- Citations
- 535
- Citations per work
- 1.8
What does Halil Önder research?
Halil Önder's work is mostly in Medicine (52%), Neuroscience (9%).
Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.
The fields and subfields, with their numbers
- Health Sciences15151.6%
- Medicine15151.6%
- Everything else11439.2%
- Life Sciences279.2%
- Neuroscience279.2%
- 1Neurology36% of their works
- 2Cellular and Molecular Neuroscience9% of their works
- 3Psychiatry and Mental health5% of their works
- 4Endocrinology, Diabetes and Metabolism4% of their works
- 5Epidemiology4% of their works
- 6Surgery3% of their works
Research topics
- Neurological disorders and treatments
- Parkinson's Disease Mechanisms and Treatments
- Cerebrospinal fluid and hydrocephalus
- Neurological and metabolic disorders
- Cerebral Venous Sinus Thrombosis
- Neurosurgical Procedures and Complications
- Traumatic Brain Injury and Neurovascular Disturbances
- Genetic Neurodegenerative Diseases
Which keywords describe Halil Önder's research?
The keywords of their largest research topics: Parkinson's Disease, Neurological Disorders, Basal Ganglia, Mitochondrial Dysfunction, Deep Brain Stimulation, Subthalamic Nucleus, a-Synuclein and Neurodegeneration.
- Protein Aggregation
- Traumatic Brain Injury
- Intracranial Hypotension
- Idiopathic Intracranial Hypertension
- Hyperglycemia
- Glymphatic System
- Intracranial Pressure
- a-Synuclein
- Subthalamic Nucleus
- Mitochondrial Dysfunction
- Neurological Disorders
- Parkinson's Disease
- Basal Ganglia
- Deep Brain Stimulation
- Neurodegeneration
- Neuroimaging
- Cerebrospinal Fluid
- Chorea
- Cerebral Venous Thrombosis
- Cerebrospinal Fluid Leaks
- Pathophysiology
- Huntington's Disease
Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.
All 22 words, with their numbers
- Parkinson's Disease1492 topics
- Neurological Disorders1262 topics
- Basal Ganglia1092 topics
- Mitochondrial Dysfunction912 topics
- Deep Brain Stimulation791 topic
- Subthalamic Nucleus791 topic
- Neurodegeneration701 topic
- a-Synuclein701 topic
- Neuroimaging592 topics
- Intracranial Pressure522 topics
- Cerebrospinal Fluid471 topic
- Glymphatic System471 topic
- Chorea301 topic
- Hyperglycemia301 topic
- Cerebral Venous Thrombosis291 topic
- Idiopathic Intracranial Hypertension291 topic
- Cerebrospinal Fluid Leaks241 topic
- Intracranial Hypotension241 topic
- Pathophysiology231 topic
- Traumatic Brain Injury231 topic
- Huntington's Disease211 topic
- Protein Aggregation211 topic
Where does Halil Önder work?
Halil Önder's main affiliation in the publication record is Dünyagöz Hospital, Türkiye.
How many publications and citations does Halil Önder have?
OpenAlex counts 292 works and 535 citations for Halil Önder, an h-index of 10, ranking #7,706,225 of 7,710,434 researchers worldwide on the quality score.
Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.
Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.
Discussion
Ask a question or share a note about Halil Önder. Keep it to the research: no personal attacks, advertising or links. Any reader can report a post, which collapses it for everyone until it is opened.