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Researcher · Regions Hospital · United States

John R. Fawcett

John R. Fawcett publishes mostly in Cellular and Molecular Neuroscience, Developmental Neuroscience and Endocrinology, Diabetes and Metabolism, on topics such as Neurogenesis and neuroplasticity mechanisms, Nerve injury and regeneration and Ophthalmology and Eye Disorders.

OpenAlex
World rank
#2,510,809
of 7,710,434 ranked researchers
Rank in United States
#739,972
of 1,627,098
Works
17
Citations
1.1k
Citations per work
64.6
h-index
8
i10-index 8Top 88% in Cellular and Molecular Neuroscience
John R. Fawcett

What does John R. Fawcett research?

John R. Fawcett's work is mostly in Neuroscience (35%), Medicine (20%), Biochemistry, Genetics and Molecular Biology (9%).

Life Sciences: 7 works · 43.5% of their workNeuroscience: 6 works · 34.8% of their workCellular and Molecular Neuroscience: 4 works · 23.9% of their workDevelopmental Neuroscience: 2 works · 10.9% of their workBiochemistry, Genetics and Molecular Biology: 1 works · 8.7% of their workMolecular Biology: 1 works · 8.7% of their workEverything else: 5 works · 30.4% of their workHealth Sciences: 3 works · 19.6% of their workMedicine: 3 works · 19.6% of their workEndocrinology, Diabetes and Metabolism: 2 works · 10.9% of their workPathology and Forensic Medicine: 1 works · 8.7% of their workSocial Sciences: 1 works · 6.5% of their workSocial Sciences: 1 works · 6.5% of their workEducation: 1 works · 6.5% of their workTHEIR WORKJohn R. Fawcett17works
Life Sciences44%Health Sciences20%Social Sciences6%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Neuroscience634.8%
    2. Biochemistry, Genetics and Molecular Biology18.7%
  2. Everything else530.4%
    1. Medicine319.6%
    1. Social Sciences16.5%
  1. 1Cellular and Molecular Neuroscience24% of their works
  2. 2Developmental Neuroscience11% of their works
  3. 3Endocrinology, Diabetes and Metabolism11% of their works
  4. 4Molecular Biology9% of their works
  5. 5Pathology and Forensic Medicine9% of their works
  6. 6Education6% of their works

Research topics

Which keywords describe John R. Fawcett's research?

The keywords of their largest research topics: Synaptic Plasticity, Adult Stem Cells, Neurogenesis, BDNF, Cell Signaling, Graves' Ophthalmopathy, Neurotrophins and Thyroid-Associated Ophthalmopathy.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 18 words, with their numbers
  1. Synaptic Plasticity72 topics
  2. Adult Stem Cells51 topic
  3. Neurogenesis51 topic
  4. BDNF41 topic
  5. Cell Signaling42 topics
  6. Graves' Ophthalmopathy41 topic
  7. Neurotrophins41 topic
  8. Thyroid-Associated Ophthalmopathy41 topic
  9. GABAergic31 topic
  10. IGF-131 topic
  11. Insulin-like Growth Factors31 topic
  12. Neurotransmission31 topic
  13. Cerebrolysin21 topic
  14. Citicoline21 topic
  15. Crystal Structure21 topic
  16. Eph receptors21 topic
  17. G-Protein-Coupled Receptors21 topic
  18. Semaphorins21 topic

Where does John R. Fawcett work?

John R. Fawcett's main affiliation in the publication record is Regions Hospital, United States.

How many publications and citations does John R. Fawcett have?

OpenAlex counts 17 works and 1,098 citations for John R. Fawcett, an h-index of 8, ranking #2,510,809 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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