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Researcher · CHDI Foundation · United States

Ladislav Mrzljak

Ladislav Mrzljak publishes mostly in Cellular and Molecular Neuroscience, Molecular Biology and Neurology, on topics such as Neuroscience and Neuropharmacology Research, Genetic Neurodegenerative Diseases and Receptor Mechanisms and Signaling.

ORCID 0000-0002-5777-1354 OpenAlex
World rank
#844,230
of 7,710,434 ranked researchers
Rank in United States
#280,260
of 1,627,098
Works
75
Citations
5.3k
Citations per work
70.8
h-index
35
i10-index 53Top 12% in Cellular and Molecular Neuroscience
Ladislav Mrzljak

What does Ladislav Mrzljak research?

Ladislav Mrzljak's work is mostly in Neuroscience (44%), Biochemistry, Genetics and Molecular Biology (25%), Medicine (11%).

Life Sciences: 52 works · 68.9% of their workNeuroscience: 33 works · 43.5% of their workCellular and Molecular Neuroscience: 26 works · 34.0% of their workCognitive Neuroscience: 5 works · 6.2% of their workDevelopmental Neuroscience: 2 works · 3.3% of their workBiochemistry, Genetics and Molecular Biology: 19 works · 25.4% of their workMolecular Biology: 19 works · 25.4% of their workEverything else: 15 works · 19.7% of their workHealth Sciences: 9 works · 11.4% of their workMedicine: 9 works · 11.4% of their workNeurology: 6 works · 8.1% of their workPediatrics, Perinatology and Child Health: 2 works · 3.3% of their workTHEIR WORKLadislav Mrzljak75works
Life Sciences69%Health Sciences11%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Neuroscience3343.5%
    2. Biochemistry, Genetics and Molecular Biology1925.4%
  2. Everything else1519.7%
    1. Medicine911.4%
  1. 1Cellular and Molecular Neuroscience34% of their works
  2. 2Molecular Biology25% of their works
  3. 3Neurology8% of their works
  4. 4Cognitive Neuroscience6% of their works
  5. 5Developmental Neuroscience3% of their works
  6. 6Pediatrics, Perinatology and Child Health3% of their works

Research topics

Which keywords describe Ladislav Mrzljak's research?

The keywords of their largest research topics: Neurological Disorders, Mitochondrial Dysfunction, Neurotransmission, Synaptic Plasticity, Crystal Structure, Huntington's Disease, Protein Aggregation and G-Protein-Coupled Receptors.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 21 words, with their numbers
  1. Neurological Disorders442 topics
  2. Mitochondrial Dysfunction302 topics
  3. Neurotransmission301 topic
  4. Synaptic Plasticity301 topic
  5. Crystal Structure262 topics
  6. Huntington's Disease201 topic
  7. Protein Aggregation201 topic
  8. G-Protein-Coupled Receptors181 topic
  9. Signal Transduction181 topic
  10. Oxidative Stress152 topics
  11. Deep Brain Stimulation141 topic
  12. Parkinson's Disease141 topic
  13. Schizophrenia132 topics
  14. Dopamine121 topic
  15. Serotonin121 topic
  16. Mitochondria101 topic
  17. Reactive Oxygen Species101 topic
  18. Neuronal Signaling81 topic
  19. Nicotinic Acetylcholine Receptors81 topic
  20. Cytokines51 topic
  21. Neuroinflammation51 topic

Where does Ladislav Mrzljak work?

Ladislav Mrzljak's main affiliation in the publication record is CHDI Foundation, United States.

How many publications and citations does Ladislav Mrzljak have?

OpenAlex counts 75 works and 5,308 citations for Ladislav Mrzljak, an h-index of 35, ranking #844,230 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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