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Researcher · Symphogen (Denmark) · Denmark

Lars Søegaard Nielsen

Lars Søegaard Nielsen publishes mostly in Hematology, Molecular Biology and Radiology, Nuclear Medicine and Imaging, on topics such as Monoclonal and Polyclonal Antibodies Research, Protease and Inhibitor Mechanisms and Blood Coagulation and Thrombosis Mechanisms.

OpenAlex
World rank
#1,773,501
of 7,710,434 ranked researchers
Rank in Denmark
#13,413
of 43,819
Works
35
Citations
1.7k
Citations per work
48.6
h-index
21
i10-index 25Top 23% in Hematology
Lars Søegaard Nielsen

What does Lars Søegaard Nielsen research?

Lars Søegaard Nielsen's work is mostly in Medicine (40%), Biochemistry, Genetics and Molecular Biology (28%), Immunology and Microbiology (6%).

Health Sciences: 14 works · 39.9% of their workMedicine: 14 works · 39.9% of their workHematology: 7 works · 19.4% of their workRadiology, Nuclear Medicine and Imaging: 5 works · 14.0% of their workImmunology and Allergy: 2 works · 6.5% of their workLife Sciences: 12 works · 34.5% of their workBiochemistry, Genetics and Molecular Biology: 10 works · 28.0% of their workMolecular Biology: 5 works · 15.1% of their workCancer Research: 5 works · 12.9% of their workImmunology and Microbiology: 2 works · 6.5% of their workImmunology: 2 works · 6.5% of their workEverything else: 9 works · 25.6% of their workTHEIR WORKLars SøegaardNielsen35works
Life Sciences34%Health Sciences40%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Medicine1439.9%
    1. Biochemistry, Genetics and Molecular Biology1028.0%
    2. Immunology and Microbiology26.5%
  3. Everything else925.6%
  1. 1Hematology19% of their works
  2. 2Molecular Biology15% of their works
  3. 3Radiology, Nuclear Medicine and Imaging14% of their works
  4. 4Cancer Research13% of their works
  5. 5Immunology and Allergy6% of their works
  6. 6Immunology6% of their works

Research topics

Which keywords describe Lars Søegaard Nielsen's research?

The keywords of their largest research topics: Angiogenesis, Extracellular Matrix, Monoclonal Antibodies, Therapeutic Antibodies, Glycosylation, Inflammation, Antibody Engineering and Fc Receptors.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 22 words, with their numbers
  1. Angiogenesis223 topics
  2. Extracellular Matrix182 topics
  3. Monoclonal Antibodies182 topics
  4. Therapeutic Antibodies182 topics
  5. Glycosylation172 topics
  6. Inflammation172 topics
  7. Antibody Engineering131 topic
  8. Fc Receptors131 topic
  9. Matrix Metalloproteinases121 topic
  10. Tissue Inhibitors of Metalloproteinases121 topic
  11. Protease-Activated Receptors111 topic
  12. Thrombin111 topic
  13. Cell Adhesion61 topic
  14. Factor VIII61 topic
  15. Hemophilia61 topic
  16. Integrins61 topic
  17. Biopharmaceuticals51 topic
  18. Protein Aggregation51 topic
  19. Fibroblast Activation Protein41 topic
  20. Mammalian Cells41 topic
  21. Recombinant Protein41 topic
  22. Tumor Microenvironment41 topic

Where does Lars Søegaard Nielsen work?

Lars Søegaard Nielsen's main affiliation in the publication record is Symphogen (Denmark), Denmark.

How many publications and citations does Lars Søegaard Nielsen have?

OpenAlex counts 35 works and 1,701 citations for Lars Søegaard Nielsen, an h-index of 21, ranking #1,773,501 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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