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Researcher · Centro de Investigaciones Cardiovasculares · Argentina

Leticia Vittone

Leticia Vittone publishes mostly in Cardiology and Cardiovascular Medicine, Molecular Biology and Pathology and Forensic Medicine, on topics such as Cardiac electrophysiology and arrhythmias, Ion channel regulation and function and Cardiac Ischemia and Reperfusion.

OpenAlex
World rank
#4,830,532
of 7,710,434 ranked researchers
Rank in Argentina
#14,706
of 38,473
Works
74
Citations
1.6k
Citations per work
21.2
h-index
22
i10-index 31Top 17% in Cardiology and Cardiovascular Medicine
Leticia Vittone

What does Leticia Vittone research?

Leticia Vittone's work is mostly in Medicine (52%), Biochemistry, Genetics and Molecular Biology (27%), Neuroscience (4%).

Health Sciences: 38 works · 52.0% of their workMedicine: 38 works · 52.0% of their workCardiology and Cardiovascular Medicine: 26 works · 35.5% of their workPathology and Forensic Medicine: 8 works · 10.5% of their workPhysiology: 2 works · 3.0% of their workRadiology, Nuclear Medicine and Imaging: 2 works · 3.0% of their workLife Sciences: 23 works · 30.5% of their workBiochemistry, Genetics and Molecular Biology: 20 works · 27.0% of their workMolecular Biology: 20 works · 27.0% of their workNeuroscience: 3 works · 3.5% of their workCellular and Molecular Neuroscience: 3 works · 3.5% of their workEverything else: 13 works · 17.5% of their workTHEIR WORKLeticia Vittone74works
Life Sciences30%Health Sciences52%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Medicine3852.0%
    1. Biochemistry, Genetics and Molecular Biology2027.0%
    2. Neuroscience33.5%
  3. Everything else1317.5%
  1. 1Cardiology and Cardiovascular Medicine36% of their works
  2. 2Molecular Biology27% of their works
  3. 3Pathology and Forensic Medicine10% of their works
  4. 4Cellular and Molecular Neuroscience4% of their works
  5. 5Physiology3% of their works
  6. 6Radiology, Nuclear Medicine and Imaging3% of their works

Research topics

Which keywords describe Leticia Vittone's research?

The keywords of their largest research topics: Ion Channels, Sudden Cardiac Death, Arrhythmias, Long-QT Syndrome, Cell Signaling, Calcium Channels, Voltage-Gated Channels and Oxidative Stress.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most.

All 23 words, with their numbers
  1. Ion Channels782 topics
  2. Sudden Cardiac Death502 topics
  3. Arrhythmias461 topic
  4. Long-QT Syndrome461 topic
  5. Cell Signaling412 topics
  6. Calcium Channels321 topic
  7. Voltage-Gated Channels321 topic
  8. Oxidative Stress252 topics
  9. Reactive Oxygen Species252 topics
  10. Cardioprotection211 topic
  11. Ischemia-Reperfusion Injury211 topic
  12. Echocardiography112 topics
  13. Heart Failure112 topics
  14. Crystal Structure91 topic
  15. G-Protein-Coupled Receptors91 topic
  16. Chamber Quantification71 topic
  17. Diastolic Function71 topic
  18. Cardiac Resynchronization Therapy41 topic
  19. Cardiomyopathies41 topic
  20. Genetic Mutations41 topic
  21. Implantable Cardioverter-Defibrillator41 topic
  22. Mitochondria41 topic
  23. Mitochondrial Dysfunction41 topic

Where does Leticia Vittone work?

Leticia Vittone's main affiliation in the publication record is Centro de Investigaciones Cardiovasculares, Argentina.

How many publications and citations does Leticia Vittone have?

OpenAlex counts 74 works and 1,569 citations for Leticia Vittone, an h-index of 22, ranking #4,830,532 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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