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Researcher · National Institutes of Health · United States

Marta Mota Vieira

Marta Mota Vieira publishes mostly in Molecular Biology, Cellular and Molecular Neuroscience and Genetics, on topics such as Neuroscience and Neuropharmacology Research, Ion channel regulation and function and Genetics and Neurodevelopmental Disorders.

ORCID 0000-0001-7332-6181 OpenAlex
World rank
#1,927,227
of 7,710,434 ranked researchers
Rank in United States
#587,722
of 1,627,098
Works
17
Citations
807
Citations per work
47.5
h-index
11
i10-index 11Top 71% in Molecular Biology
Marta Mota Vieira

What does Marta Mota Vieira research?

Marta Mota Vieira's work is mostly in Biochemistry, Genetics and Molecular Biology (46%), Neuroscience (35%), Medicine (4%).

Life Sciences: 14 works · 82.7% of their workBiochemistry, Genetics and Molecular Biology: 8 works · 45.7% of their workMolecular Biology: 6 works · 37.0% of their workGenetics: 1 works · 8.7% of their workNeuroscience: 6 works · 34.8% of their workCellular and Molecular Neuroscience: 4 works · 26.1% of their workNeurology: 1 works · 8.7% of their workAgricultural and Biological Sciences: 0 works · 2.2% of their workAnimal Science and Zoology: 0 works · 2.2% of their workEverything else: 2 works · 13.0% of their workHealth Sciences: 1 works · 4.3% of their workMedicine: 1 works · 4.3% of their workBiochemistry: 1 works · 4.3% of their workTHEIR WORKMarta Mota Vieira17works
Life Sciences83%Health Sciences4%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology845.7%
    2. Neuroscience634.8%
    3. Agricultural and Biological Sciences02.2%
  2. Everything else213.0%
    1. Medicine14.3%
  1. 1Molecular Biology37% of their works
  2. 2Cellular and Molecular Neuroscience26% of their works
  3. 3Genetics9% of their works
  4. 4Neurology9% of their works
  5. 5Biochemistry4% of their works
  6. 6Animal Science and Zoology2% of their works

Research topics

Which keywords describe Marta Mota Vieira's research?

The keywords of their largest research topics: Dendritic Spines, Neurotransmission, Synaptic Plasticity, Cell Signaling, Brain Development, Ion Channels, Voltage-Gated Channels and Cell Death.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 21 words, with their numbers
  1. Dendritic Spines152 topics
  2. Neurotransmission111 topic
  3. Synaptic Plasticity111 topic
  4. Cell Signaling82 topics
  5. Brain Development72 topics
  6. Ion Channels51 topic
  7. Voltage-Gated Channels51 topic
  8. Cell Death42 topics
  9. Fragile X Syndrome41 topic
  10. Mitochondria42 topics
  11. Rett Syndrome41 topic
  12. Crystal Structure31 topic
  13. G-Protein-Coupled Receptors31 topic
  14. Microglia31 topic
  15. Neuroinflammation31 topic
  16. Apoptosis21 topic
  17. Bcl-2 Family21 topic
  18. Mitochondrial Dysfunction21 topic
  19. Oxidative Stress21 topic
  20. Meat Quality11 topic
  21. Protein Oxidation11 topic

Where does Marta Mota Vieira work?

Marta Mota Vieira's main affiliation in the publication record is National Institutes of Health, United States.

How many publications and citations does Marta Mota Vieira have?

OpenAlex counts 17 works and 807 citations for Marta Mota Vieira, an h-index of 11, ranking #1,927,227 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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