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Researcher · German Center for Neurodegenerative Diseases · Germany

Mikael Simons

Mikael Simons publishes mostly in Molecular Biology, Neurology and Developmental Neuroscience, on topics such as Neuroinflammation and Neurodegeneration Mechanisms, Neurogenesis and neuroplasticity mechanisms and Alzheimer's disease research and treatments.

ORCID 0000-0001-5329-192X OpenAlex
World rank
#8,593
of 7,710,434 ranked researchers
Rank in Germany
#379
of 364,343
Works
234
Citations
37k
Citations per work
159.5
h-index
88
i10-index 169Top 0.4% in Molecular Biology
Mikael Simons

What does Mikael Simons research?

Mikael Simons's work is mostly in Neuroscience (29%), Biochemistry, Genetics and Molecular Biology (22%), Immunology and Microbiology (10%).

Life Sciences: 141 works · 60.4% of their workNeuroscience: 67 works · 28.8% of their workNeurology: 29 works · 12.6% of their workDevelopmental Neuroscience: 24 works · 10.1% of their workCellular and Molecular Neuroscience: 14 works · 6.1% of their workBiochemistry, Genetics and Molecular Biology: 52 works · 22.1% of their workMolecular Biology: 52 works · 22.1% of their workImmunology and Microbiology: 22 works · 9.5% of their workImmunology: 22 works · 9.5% of their workEverything else: 71 works · 30.5% of their workHealth Sciences: 21 works · 9.1% of their workMedicine: 21 works · 9.1% of their workPhysiology: 21 works · 9.1% of their workTHEIR WORKMikael Simons234works
Life Sciences60%Health Sciences9%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Neuroscience6728.8%
    2. Biochemistry, Genetics and Molecular Biology5222.1%
    3. Immunology and Microbiology229.5%
  2. Everything else7130.5%
    1. Medicine219.1%
  1. 1Molecular Biology22% of their works
  2. 2Neurology13% of their works
  3. 3Developmental Neuroscience10% of their works
  4. 4Immunology10% of their works
  5. 5Physiology9% of their works
  6. 6Cellular and Molecular Neuroscience6% of their works

Research topics

Which keywords describe Mikael Simons's research?

The keywords of their largest research topics: Brain Development, Neurodegeneration, Neuroinflammation, Astrocytes, Microglia, Adult Stem Cells, Neurogenesis and Cancer.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 22 words, with their numbers
  1. Brain Development1342 topics
  2. Neurodegeneration1212 topics
  3. Neuroinflammation1212 topics
  4. Astrocytes681 topic
  5. Microglia681 topic
  6. Adult Stem Cells661 topic
  7. Neurogenesis661 topic
  8. Cancer573 topics
  9. Amyloid Hypothesis531 topic
  10. Tau Pathology531 topic
  11. Biogenesis312 topics
  12. Biomarkers312 topics
  13. Activation261 topic
  14. Macrophage261 topic
  15. Exosomes171 topic
  16. Extracellular Vesicles171 topic
  17. Lipid Rafts171 topic
  18. Membrane Lipids171 topic
  19. BDNF161 topic
  20. Neurotrophins161 topic
  21. Gene Expression141 topic
  22. MicroRNA141 topic

Where does Mikael Simons work?

Mikael Simons's main affiliation in the publication record is German Center for Neurodegenerative Diseases, Germany.

How many publications and citations does Mikael Simons have?

OpenAlex counts 234 works and 37,331 citations for Mikael Simons, an h-index of 88, ranking #8,593 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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