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Researcher · Instituto de Neurología de Buenos Aires · Argentina

Natalia González Rojas

Natalia González Rojas publishes mostly in Neurology, Cellular and Molecular Neuroscience and Genetics, on topics such as Neurological disorders and treatments, Genetic Neurodegenerative Diseases and Parkinson's Disease Mechanisms and Treatments.

ORCID 0000-0002-6273-2683 OpenAlex
World rank
#4,781,995
of 7,710,434 ranked researchers
Rank in Argentina
#14,464
of 38,473
Works
22
Citations
187
Citations per work
8.5
h-index
5
i10-index 4Top 90% in Neurology (Medicine)
Natalia González Rojas

What does Natalia González Rojas research?

Natalia González Rojas's work is mostly in Medicine (50%), Biochemistry, Genetics and Molecular Biology (14%), Neuroscience (12%).

Health Sciences: 11 works · 50.1% of their workMedicine: 11 works · 50.1% of their workNeurology: 9 works · 39.7% of their workEndocrinology, Diabetes and Metabolism: 1 works · 5.2% of their workPsychiatry and Mental health: 1 works · 5.2% of their workLife Sciences: 6 works · 25.9% of their workBiochemistry, Genetics and Molecular Biology: 3 works · 13.8% of their workGenetics: 2 works · 8.6% of their workMolecular Biology: 1 works · 5.2% of their workNeuroscience: 3 works · 12.1% of their workCellular and Molecular Neuroscience: 3 works · 12.1% of their workEverything else: 5 works · 24.0% of their workTHEIR WORKNatalia GonzálezRojas22works
Life Sciences26%Health Sciences50%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Medicine1150.1%
    1. Biochemistry, Genetics and Molecular Biology313.8%
    2. Neuroscience312.1%
  3. Everything else524.0%
  1. 1Neurology40% of their works
  2. 2Cellular and Molecular Neuroscience12% of their works
  3. 3Genetics9% of their works
  4. 4Psychiatry and Mental health5% of their works
  5. 5Molecular Biology5% of their works
  6. 6Endocrinology, Diabetes and Metabolism5% of their works

Research topics

Which keywords describe Natalia González Rojas's research?

The keywords of their largest research topics: Mitochondrial Dysfunction, Parkinson's Disease, Basal Ganglia, Neurodegeneration, Deep Brain Stimulation, Oxidative Stress, Subthalamic Nucleus and a-Synuclein.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 21 words, with their numbers
  1. Mitochondrial Dysfunction163 topics
  2. Parkinson's Disease162 topics
  3. Basal Ganglia122 topics
  4. Neurodegeneration102 topics
  5. Deep Brain Stimulation91 topic
  6. Oxidative Stress92 topics
  7. Subthalamic Nucleus91 topic
  8. Dopaminergic Neurons71 topic
  9. Huntington's Disease71 topic
  10. Protein Aggregation71 topic
  11. a-Synuclein71 topic
  12. Chorea31 topic
  13. FUS/TLS31 topic
  14. Genetic Variants31 topic
  15. Hyperglycemia31 topic
  16. Sequence Interpretation31 topic
  17. TDP-4331 topic
  18. Biochemical Genetics21 topic
  19. Metabolic Disorders21 topic
  20. Mitochondria21 topic
  21. Reactive Oxygen Species21 topic

Where does Natalia González Rojas work?

Natalia González Rojas's main affiliation in the publication record is Instituto de Neurología de Buenos Aires, Argentina.

How many publications and citations does Natalia González Rojas have?

OpenAlex counts 22 works and 187 citations for Natalia González Rojas, an h-index of 5, ranking #4,781,995 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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