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Researcher · Nordic Bioscience (Denmark) · Denmark

Nicholas Willumsen

Nicholas Willumsen publishes mostly in Oncology, Molecular Biology and Cancer Research, on topics such as Cell Adhesion Molecules Research, Protease and Inhibitor Mechanisms and Pancreatic and Hepatic Oncology Research.

ORCID 0000-0002-5207-5173 OpenAlex
World rank
#665,619
of 7,710,434 ranked researchers
Rank in Denmark
#4,994
of 43,819
Works
208
Citations
3k
Citations per work
14.6
h-index
33
i10-index 60Top 9.4% in Oncology
Nicholas Willumsen

What does Nicholas Willumsen research?

Nicholas Willumsen's work is mostly in Medicine (42%), Biochemistry, Genetics and Molecular Biology (22%), Immunology and Microbiology (5%).

Health Sciences: 88 works · 42.4% of their workMedicine: 88 works · 42.4% of their workOncology: 52 works · 25.2% of their workImmunology and Allergy: 21 works · 10.3% of their workPulmonary and Respiratory Medicine: 14 works · 6.9% of their workEverything else: 64 works · 30.8% of their workLife Sciences: 56 works · 26.8% of their workBiochemistry, Genetics and Molecular Biology: 45 works · 21.6% of their workMolecular Biology: 23 works · 11.1% of their workCancer Research: 22 works · 10.5% of their workImmunology and Microbiology: 11 works · 5.2% of their workImmunology: 11 works · 5.2% of their workTHEIR WORKNicholasWillumsen208works
Life Sciences27%Health Sciences42%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Medicine8842.4%
  2. Everything else6430.8%
    1. Biochemistry, Genetics and Molecular Biology4521.6%
    2. Immunology and Microbiology115.2%
  1. 1Oncology25% of their works
  2. 2Molecular Biology11% of their works
  3. 3Cancer Research10% of their works
  4. 4Immunology and Allergy10% of their works
  5. 5Pulmonary and Respiratory Medicine7% of their works
  6. 6Immunology5% of their works

Research topics

Which keywords describe Nicholas Willumsen's research?

The keywords of their largest research topics: Angiogenesis, Extracellular Matrix, Tumor Microenvironment, Cancer Progression, Metastasis, Cell Adhesion, Integrins and Matrix Metalloproteinases.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 22 words, with their numbers
  1. Angiogenesis1454 topics
  2. Extracellular Matrix1042 topics
  3. Tumor Microenvironment663 topics
  4. Cancer Progression652 topics
  5. Metastasis652 topics
  6. Cell Adhesion561 topic
  7. Integrins561 topic
  8. Matrix Metalloproteinases481 topic
  9. Tissue Inhibitors of Metalloproteinases481 topic
  10. Chemotherapy382 topics
  11. Pancreatic Cancer251 topic
  12. Surgical Complications251 topic
  13. Cancer Stem Cells241 topic
  14. Fibroblast Activation Protein241 topic
  15. Immunosuppression241 topic
  16. Tumor Metastasis241 topic
  17. Immune Checkpoint Blockade171 topic
  18. PD-1 and PD-L1171 topic
  19. Smad-dependent pathways171 topic
  20. Tumor Suppression171 topic
  21. Cetuximab131 topic
  22. KRAS Mutations131 topic

Where does Nicholas Willumsen work?

Nicholas Willumsen's main affiliation in the publication record is Nordic Bioscience (Denmark), Denmark.

How many publications and citations does Nicholas Willumsen have?

OpenAlex counts 208 works and 3,046 citations for Nicholas Willumsen, an h-index of 33, ranking #665,619 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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