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Researcher · Hamad bin Khalifa University · Qatar

Omar M. A. El‐Agnaf

Omar M. A. El‐Agnaf publishes mostly in Neurology, Physiology and Molecular Biology, on topics such as Parkinson's Disease Mechanisms and Treatments, Alzheimer's disease research and treatments and Neurological disorders and treatments.

ORCID 0000-0002-6850-8084 OpenAlex
World rank
#80,266
of 7,710,434 ranked researchers
Rank in Qatar
#74
of 3,423
Works
210
Citations
20k
Citations per work
96.4
h-index
64
i10-index 141Top 1.5% in Neurology (Medicine)
Omar M. A. El‐Agnaf

What does Omar M. A. El‐Agnaf research?

Omar M. A. El‐Agnaf's work is mostly in Medicine (51%), Biochemistry, Genetics and Molecular Biology (15%), Neuroscience (12%).

Health Sciences: 106 works · 50.7% of their workMedicine: 106 works · 50.7% of their workNeurology: 74 works · 35.3% of their workPhysiology: 32 works · 15.4% of their workLife Sciences: 56 works · 26.7% of their workBiochemistry, Genetics and Molecular Biology: 31 works · 14.6% of their workMolecular Biology: 25 works · 12.1% of their workCell Biology: 5 works · 2.5% of their workNeuroscience: 25 works · 12.1% of their workCellular and Molecular Neuroscience: 17 works · 8.0% of their workNeurology: 9 works · 4.1% of their workEverything else: 47 works · 22.6% of their workTHEIR WORKOmar M. A.El‐Agnaf210works
Life Sciences27%Health Sciences51%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Medicine10650.7%
    1. Biochemistry, Genetics and Molecular Biology3114.6%
    2. Neuroscience2512.1%
  3. Everything else4722.6%
  1. 1Neurology35% of their works
  2. 2Physiology15% of their works
  3. 3Molecular Biology12% of their works
  4. 4Cellular and Molecular Neuroscience8% of their works
  5. 5Neurology4% of their works
  6. 6Cell Biology2% of their works

Research topics

Which keywords describe Omar M. A. El‐Agnaf's research?

The keywords of their largest research topics: Neurodegeneration, Oxidative Stress, Parkinson's Disease, Dopaminergic Neurons, Mitochondrial Dysfunction, a-Synuclein, Genetic Risk Factors and Amyloid Hypothesis.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 23 words, with their numbers
  1. Neurodegeneration2303 topics
  2. Oxidative Stress2062 topics
  3. Parkinson's Disease2063 topics
  4. Dopaminergic Neurons1512 topics
  5. Mitochondrial Dysfunction1362 topics
  6. Genetic Risk Factors1271 topic
  7. a-Synuclein1271 topic
  8. Amyloid Hypothesis791 topic
  9. Tau Pathology791 topic
  10. Neurological Disorders702 topics
  11. Deep Brain Stimulation551 topic
  12. Subthalamic Nucleus551 topic
  13. NR4A2241 topic
  14. Nurr1241 topic
  15. Protein Aggregation182 topics
  16. Botulinum Toxin151 topic
  17. Neurotoxins151 topic
  18. Ginkgo Biloba91 topic
  19. Huntington's Disease91 topic
  20. Molecular Basis91 topic
  21. Neuroprotective91 topic
  22. Polyglutamine91 topic
  23. Prion Diseases91 topic

Where does Omar M. A. El‐Agnaf work?

Omar M. A. El‐Agnaf's main affiliation in the publication record is Hamad bin Khalifa University, Qatar.

How many publications and citations does Omar M. A. El‐Agnaf have?

OpenAlex counts 210 works and 20,253 citations for Omar M. A. El‐Agnaf, an h-index of 64, ranking #80,266 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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