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Researcher · Boston Biomedical Research Institute · United States

Paul C. Leavis

Paul C. Leavis publishes mostly in Molecular Biology, Cardiology and Cardiovascular Medicine and Cell Biology, on topics such as Cardiomyopathy and Myosin Studies, Mass Spectrometry Techniques and Applications and RNA and protein synthesis mechanisms.

OpenAlex
World rank
#907,315
of 7,710,434 ranked researchers
Rank in United States
#299,105
of 1,627,098
Works
71
Citations
3.6k
Citations per work
51.1
h-index
35
i10-index 50Top 10% in Molecular Biology
Paul C. Leavis

What does Paul C. Leavis research?

Paul C. Leavis's work is mostly in Biochemistry, Genetics and Molecular Biology (33%), Medicine (18%), Immunology and Microbiology (5%).

Life Sciences: 27 works · 38.1% of their workBiochemistry, Genetics and Molecular Biology: 23 works · 32.9% of their workMolecular Biology: 19 works · 26.6% of their workCell Biology: 4 works · 6.3% of their workImmunology and Microbiology: 4 works · 5.2% of their workImmunology: 4 works · 5.2% of their workEverything else: 26 works · 36.4% of their workHealth Sciences: 13 works · 17.7% of their workMedicine: 13 works · 17.7% of their workCardiology and Cardiovascular Medicine: 13 works · 17.7% of their workPhysical Sciences: 6 works · 7.8% of their workChemistry: 4 works · 5.2% of their workSpectroscopy: 4 works · 5.2% of their workPhysics and Astronomy: 2 works · 2.6% of their workAtomic and Molecular Physics, and Optics: 2 works · 2.6% of their workTHEIR WORKPaul C. Leavis71works
Life Sciences38%Physical Sciences8%Health Sciences18%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology2332.9%
    2. Immunology and Microbiology45.2%
  2. Everything else2636.4%
    1. Medicine1317.7%
    1. Chemistry45.2%
    2. Physics and Astronomy22.6%
  1. 1Molecular Biology27% of their works
  2. 2Cardiology and Cardiovascular Medicine18% of their works
  3. 3Cell Biology6% of their works
  4. 4Spectroscopy5% of their works
  5. 5Immunology5% of their works
  6. 6Atomic and Molecular Physics, and Optics3% of their works

Research topics

Which keywords describe Paul C. Leavis's research?

The keywords of their largest research topics: Genetic Mutations, Cardiomyopathies, Myosin, Ionization, Mass Spectrometry, Decidual NK Cells, Regulatory T Cells and Ribosome.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 17 words, with their numbers
  1. Genetic Mutations232 topics
  2. Cardiomyopathies171 topic
  3. Myosin171 topic
  4. Ionization101 topic
  5. Mass Spectrometry101 topic
  6. Decidual NK Cells91 topic
  7. Regulatory T Cells91 topic
  8. Ribosome91 topic
  9. Translation91 topic
  10. Ion Channels81 topic
  11. Voltage-Gated Channels81 topic
  12. Arrhythmogenic Right Ventricular Cardiomyopathy61 topic
  13. Sudden Death61 topic
  14. Alternative Splicing51 topic
  15. Cell Mechanics51 topic
  16. Extracellular Matrix51 topic
  17. RNA-Binding Proteins51 topic

Where does Paul C. Leavis work?

Paul C. Leavis's main affiliation in the publication record is Boston Biomedical Research Institute, United States.

How many publications and citations does Paul C. Leavis have?

OpenAlex counts 71 works and 3,630 citations for Paul C. Leavis, an h-index of 35, ranking #907,315 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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