Poul Henning Jensen
Poul Henning Jensen publishes mostly in Neurology, Cellular and Molecular Neuroscience and Molecular Biology, on topics such as Parkinson's Disease Mechanisms and Treatments, Neurological disorders and treatments and Alzheimer's disease research and treatments.
- World rank
- #168,633 of 7,710,434 ranked researchers
- Rank in Denmark
- #1,106 of 43,819
- Works
- 259
- Citations
- 14k
- Citations per work
- 54.3
What does Poul Henning Jensen research?
Poul Henning Jensen's work is mostly in Medicine (39%), Biochemistry, Genetics and Molecular Biology (14%), Neuroscience (12%).
Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.
The fields and subfields, with their numbers
- Health Sciences10239.3%
- Medicine10239.3%
- Life Sciences8030.9%
- Everything else7729.8%
- 1Neurology31% of their works
- 2Cellular and Molecular Neuroscience12% of their works
- 3Molecular Biology11% of their works
- 4Physiology8% of their works
- 5Plant Science5% of their works
- 6Genetics4% of their works
Research topics
- Parkinson's Disease Mechanisms and Treatments
- Neurological disorders and treatments
- Alzheimer's disease research and treatments
- Nuclear Receptors and Signaling
- Nerve injury and regeneration
- Genetic Neurodegenerative Diseases
- Botulinum Toxin and Related Neurological Disorders
- Neurological diseases and metabolism
Which keywords describe Poul Henning Jensen's research?
The keywords of their largest research topics: Neurodegeneration, Parkinson's Disease, Oxidative Stress, Mitochondrial Dysfunction, Dopaminergic Neurons, a-Synuclein, Genetic Risk Factors and Neurological Disorders.
- Pantothenate Kinase
- Neurotoxins
- Protein Aggregation
- Neurotrophins
- Nurr1
- Tau Pathology
- Subthalamic Nucleus
- Neurological Disorders
- Genetic Risk Factors
- Mitochondrial Dysfunction
- Parkinson's Disease
- Neurodegeneration
- Oxidative Stress
- Dopaminergic Neurons
- a-Synuclein
- Deep Brain Stimulation
- Amyloid Hypothesis
- NR4A2
- BDNF
- Huntington's Disease
- Botulinum Toxin
- Brain Iron Accumulation
Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.
All 22 words, with their numbers
- Neurodegeneration2384 topics
- Parkinson's Disease2293 topics
- Oxidative Stress1962 topics
- Mitochondrial Dysfunction1773 topics
- Dopaminergic Neurons1712 topics
- Genetic Risk Factors1431 topic
- a-Synuclein1431 topic
- Neurological Disorders752 topics
- Deep Brain Stimulation581 topic
- Subthalamic Nucleus581 topic
- Amyloid Hypothesis531 topic
- Tau Pathology531 topic
- NR4A2281 topic
- Nurr1281 topic
- BDNF221 topic
- Neurotrophins221 topic
- Huntington's Disease201 topic
- Protein Aggregation201 topic
- Botulinum Toxin171 topic
- Neurotoxins171 topic
- Brain Iron Accumulation141 topic
- Pantothenate Kinase141 topic
Where does Poul Henning Jensen work?
Poul Henning Jensen's main affiliation in the publication record is Aarhus University, Denmark.
How many publications and citations does Poul Henning Jensen have?
OpenAlex counts 259 works and 14,056 citations for Poul Henning Jensen, an h-index of 61, ranking #168,633 of 7,710,434 researchers worldwide on the quality score.
Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.
Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.
Discussion
Ask a question or share a note about Poul Henning Jensen. Keep it to the research: no personal attacks, advertising or links. Any reader can report a post, which collapses it for everyone until it is opened.