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Researcher · Duke Medical Center · United States

Robert J. Lefkowitz

Robert J. Lefkowitz publishes mostly in Molecular Biology, Cellular and Molecular Neuroscience and Animal Science and Zoology, on topics such as Receptor Mechanisms and Signaling, Neuropeptides and Animal Physiology and Protein Kinase Regulation and GTPase Signaling.

ORCID 0000-0003-1472-7545 OpenAlex
World rank
#188
of 7,710,434 ranked researchers
Rank in United States
#100
of 1,627,098
Works
857
Citations
148k
Citations per work
172.9
h-index
214
i10-index 689Top 0.1% in Molecular Biology
Robert J. Lefkowitz

What does Robert J. Lefkowitz research?

Robert J. Lefkowitz's work is mostly in Biochemistry, Genetics and Molecular Biology (44%), Neuroscience (18%), Medicine (8%).

Life Sciences: 583 works · 68.0% of their workBiochemistry, Genetics and Molecular Biology: 381 works · 44.5% of their workMolecular Biology: 381 works · 44.5% of their workNeuroscience: 151 works · 17.6% of their workCellular and Molecular Neuroscience: 151 works · 17.6% of their workAgricultural and Biological Sciences: 51 works · 5.9% of their workAnimal Science and Zoology: 51 works · 5.9% of their workEverything else: 189 works · 22.0% of their workHealth Sciences: 65 works · 7.6% of their workMedicine: 65 works · 7.6% of their workPhysiology: 36 works · 4.2% of their workCardiology and Cardiovascular Medicine: 29 works · 3.4% of their workPhysical Sciences: 21 works · 2.4% of their workChemistry: 21 works · 2.4% of their workSpectroscopy: 21 works · 2.4% of their workTHEIR WORKRobert J.Lefkowitz857works
Life Sciences68%Physical Sciences2%Health Sciences8%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology38144.5%
    2. Neuroscience15117.6%
    3. Agricultural and Biological Sciences515.9%
  2. Everything else18922.0%
    1. Medicine657.6%
    1. Chemistry212.4%
  1. 1Molecular Biology44% of their works
  2. 2Cellular and Molecular Neuroscience18% of their works
  3. 3Animal Science and Zoology6% of their works
  4. 4Physiology4% of their works
  5. 5Cardiology and Cardiovascular Medicine3% of their works
  6. 6Spectroscopy2% of their works

Research topics

Which keywords describe Robert J. Lefkowitz's research?

The keywords of their largest research topics: Cell Signaling, Crystal Structure, G-Protein-Coupled Receptors, Neuropeptides, Receptors, Protein Kinases, Ras Signaling and Clenbuterol.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most.

All 18 words, with their numbers
  1. Cell Signaling7322 topics
  2. Crystal Structure6821 topic
  3. G-Protein-Coupled Receptors6821 topic
  4. Neuropeptides3031 topic
  5. Receptors3031 topic
  6. Protein Kinases1471 topic
  7. Ras Signaling1471 topic
  8. Clenbuterol1431 topic
  9. Ractopamine1431 topic
  10. Membrane Proteins982 topics
  11. Neurotransmission681 topic
  12. Synaptic Plasticity681 topic
  13. Ion Channels501 topic
  14. Voltage-Gated Channels501 topic
  15. Lipid Rafts481 topic
  16. Membrane Lipids481 topic
  17. Ionization411 topic
  18. Mass Spectrometry411 topic

Where does Robert J. Lefkowitz work?

Robert J. Lefkowitz's main affiliation in the publication record is Duke Medical Center, United States.

How many publications and citations does Robert J. Lefkowitz have?

OpenAlex counts 857 works and 148,168 citations for Robert J. Lefkowitz, an h-index of 214, ranking #188 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Other researchers named Robert J. Lefkowitz

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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