Sidney Strickland
Sidney Strickland publishes mostly in Molecular Biology, Cancer Research and Physiology, on topics such as Protease and Inhibitor Mechanisms, Alzheimer's disease research and treatments and Blood Coagulation and Thrombosis Mechanisms.
- World rank
- #55,172 of 7,710,434 ranked researchers
- Rank in United States
- #21,308 of 1,627,098
- Works
- 234
- Citations
- 23k
- Citations per work
- 100.1
What does Sidney Strickland research?
Sidney Strickland's work is mostly in Biochemistry, Genetics and Molecular Biology (30%), Medicine (14%), Neuroscience (13%).
Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.
The fields and subfields, with their numbers
- Everything else10243.8%
- Life Sciences10042.7%
- Biochemistry, Genetics and Molecular Biology7030.1%
- Neuroscience2912.6%
- Health Sciences3213.5%
- Medicine3213.5%
- 1Molecular Biology21% of their works
- 2Cancer Research9% of their works
- 3Physiology8% of their works
- 4Cellular and Molecular Neuroscience7% of their works
- 5Neurology6% of their works
- 6Hematology6% of their works
Research topics
- Protease and Inhibitor Mechanisms
- Alzheimer's disease research and treatments
- Blood Coagulation and Thrombosis Mechanisms
- Coagulation, Bradykinin, Polyphosphates, and Angioedema
- RNA Research and Splicing
- Cell Adhesion Molecules Research
- Blood properties and coagulation
- Neuroinflammation and Neurodegeneration Mechanisms
Which keywords describe Sidney Strickland's research?
The keywords of their largest research topics: Angiogenesis, Extracellular Matrix, Neurodegeneration, Neuroinflammation, Matrix Metalloproteinases, Tissue Inhibitors of Metalloproteinases, Inflammation and Hemostasis.
- Microglia
- Transglutaminases
- Integrins
- RNA-Binding Proteins
- Kinin Receptor Family
- Thrombin
- Tau Pathology
- Hemostasis
- Tissue Inhibitors of Metalloproteinases
- Neuroinflammation
- Extracellular Matrix
- Angiogenesis
- Neurodegeneration
- Matrix Metalloproteinases
- Inflammation
- Amyloid Hypothesis
- Protease-Activated Receptors
- Hereditary Angioedema
- Alternative Splicing
- Cell Adhesion
- Fibrinogen
- Astrocytes
Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.
All 22 words, with their numbers
- Angiogenesis782 topics
- Extracellular Matrix782 topics
- Neurodegeneration622 topics
- Neuroinflammation622 topics
- Matrix Metalloproteinases581 topic
- Tissue Inhibitors of Metalloproteinases581 topic
- Inflammation572 topics
- Hemostasis562 topics
- Amyloid Hypothesis441 topic
- Tau Pathology441 topic
- Protease-Activated Receptors371 topic
- Thrombin371 topic
- Hereditary Angioedema231 topic
- Kinin Receptor Family231 topic
- Alternative Splicing211 topic
- RNA-Binding Proteins211 topic
- Cell Adhesion201 topic
- Integrins201 topic
- Fibrinogen191 topic
- Transglutaminases191 topic
- Astrocytes181 topic
- Microglia181 topic
Where does Sidney Strickland work?
Sidney Strickland's main affiliation in the publication record is Rockefeller University, United States.
How many publications and citations does Sidney Strickland have?
OpenAlex counts 234 works and 23,426 citations for Sidney Strickland, an h-index of 83, ranking #55,172 of 7,710,434 researchers worldwide on the quality score.
Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.
Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.
Discussion
Ask a question or share a note about Sidney Strickland. Keep it to the research: no personal attacks, advertising or links. Any reader can report a post, which collapses it for everyone until it is opened.