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Researcher · Temple University · United States

Steven R. Houser

Steven R. Houser publishes mostly in Cardiology and Cardiovascular Medicine, Molecular Biology and Surgery, on topics such as Cardiac electrophysiology and arrhythmias, Ion channel regulation and function and Cardiac Fibrosis and Remodeling.

ORCID 0000-0001-6359-5425 OpenAlex
World rank
#72,253
of 7,710,434 ranked researchers
Rank in United States
#27,887
of 1,627,098
Works
437
Citations
21k
Citations per work
48.4
h-index
81
i10-index 230Top 0.8% in Cardiology and Cardiovascular Medicine
Steven R. Houser

What does Steven R. Houser research?

Steven R. Houser's work is mostly in Medicine (50%), Biochemistry, Genetics and Molecular Biology (25%), Neuroscience (3%).

Health Sciences: 218 works · 50.0% of their workMedicine: 218 works · 50.0% of their workCardiology and Cardiovascular Medicine: 164 works · 37.6% of their workSurgery: 28 works · 6.5% of their workPathology and Forensic Medicine: 15 works · 3.5% of their workPhysiology: 10 works · 2.4% of their workLife Sciences: 121 works · 27.8% of their workBiochemistry, Genetics and Molecular Biology: 108 works · 24.7% of their workMolecular Biology: 108 works · 24.7% of their workNeuroscience: 14 works · 3.1% of their workCellular and Molecular Neuroscience: 14 works · 3.1% of their workEverything else: 97 works · 22.2% of their workTHEIR WORKSteven R. Houser437works
Life Sciences28%Health Sciences50%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Medicine21850.0%
    1. Biochemistry, Genetics and Molecular Biology10824.7%
    2. Neuroscience143.1%
  3. Everything else9722.2%
  1. 1Cardiology and Cardiovascular Medicine38% of their works
  2. 2Molecular Biology25% of their works
  3. 3Surgery6% of their works
  4. 4Pathology and Forensic Medicine4% of their works
  5. 5Cellular and Molecular Neuroscience3% of their works
  6. 6Physiology2% of their works

Research topics

Which keywords describe Steven R. Houser's research?

The keywords of their largest research topics: Ion Channels, Arrhythmias, Long-QT Syndrome, Heart Failure, Extracellular Matrix, Myocardial Infarction, Calcium Channels and Voltage-Gated Channels.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 20 words, with their numbers
  1. Ion Channels2252 topics
  2. Arrhythmias1421 topic
  3. Long-QT Syndrome1421 topic
  4. Heart Failure1382 topics
  5. Extracellular Matrix1202 topics
  6. Myocardial Infarction1122 topics
  7. Calcium Channels831 topic
  8. Voltage-Gated Channels831 topic
  9. Cardiac Remodeling731 topic
  10. Inflammatory Response731 topic
  11. Chamber Quantification651 topic
  12. Echocardiography651 topic
  13. Decellularization471 topic
  14. Organ Engineering471 topic
  15. Cardiomyopathies441 topic
  16. Genetic Mutations441 topic
  17. Cardiomyocyte Renewal431 topic
  18. Heart Regeneration431 topic
  19. Cardioprotection391 topic
  20. Ischemia-Reperfusion Injury391 topic

Where does Steven R. Houser work?

Steven R. Houser's main affiliation in the publication record is Temple University, United States.

How many publications and citations does Steven R. Houser have?

OpenAlex counts 437 works and 21,131 citations for Steven R. Houser, an h-index of 81, ranking #72,253 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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