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Researcher · National Chemical Laboratory · India

Subashchandrabose Chinnathambi

Subashchandrabose Chinnathambi publishes mostly in Physiology, Molecular Biology and Neurology, on topics such as Alzheimer's disease research and treatments, Neuroinflammation and Neurodegeneration Mechanisms and Cholinesterase and Neurodegenerative Diseases.

ORCID 0000-0002-5468-2129 OpenAlex
World rank
#23,532
of 7,710,434 ranked researchers
Rank in India
#245
of 280,097
Works
176
Citations
5.4k
Citations per work
30.4
h-index
46
i10-index 86Top 4.6% in Physiology (Medicine)
Subashchandrabose Chinnathambi

What does Subashchandrabose Chinnathambi research?

Subashchandrabose Chinnathambi's work is mostly in Medicine (27%), Biochemistry, Genetics and Molecular Biology (24%), Neuroscience (17%).

Life Sciences: 72 works · 41.0% of their workBiochemistry, Genetics and Molecular Biology: 42 works · 23.8% of their workMolecular Biology: 31 works · 17.4% of their workCell Biology: 11 works · 6.4% of their workNeuroscience: 30 works · 17.2% of their workNeurology: 19 works · 10.8% of their workCellular and Molecular Neuroscience: 11 works · 6.4% of their workEverything else: 57 works · 32.2% of their workHealth Sciences: 47 works · 26.8% of their workMedicine: 47 works · 26.8% of their workPhysiology: 38 works · 21.8% of their workPharmacology: 9 works · 5.0% of their workTHEIR WORKSubashchandraboseChinnathambi176works
Life Sciences41%Health Sciences27%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology4223.8%
    2. Neuroscience3017.2%
  2. Everything else5732.2%
    1. Medicine4726.8%
  1. 1Physiology22% of their works
  2. 2Molecular Biology17% of their works
  3. 3Neurology11% of their works
  4. 4Cell Biology6% of their works
  5. 5Cellular and Molecular Neuroscience6% of their works
  6. 6Pharmacology5% of their works

Research topics

Which keywords describe Subashchandrabose Chinnathambi's research?

The keywords of their largest research topics: Neurodegeneration, Neuroinflammation, Amyloid Hypothesis, Tau Pathology, Astrocytes, Microglia, Neurodegenerative Diseases and Cancer Therapy.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 20 words, with their numbers
  1. Neurodegeneration1562 topics
  2. Neuroinflammation1562 topics
  3. Amyloid Hypothesis1121 topic
  4. Tau Pathology1121 topic
  5. Astrocytes441 topic
  6. Microglia441 topic
  7. Neurodegenerative Diseases332 topics
  8. Cancer Therapy262 topics
  9. Acetylcholinesterase211 topic
  10. Cholinesterase Inhibitors211 topic
  11. Neurotransmission141 topic
  12. Synaptic Plasticity141 topic
  13. Cell Division131 topic
  14. HDAC Inhibitors131 topic
  15. Histone Deacetylases131 topic
  16. Microtubules131 topic
  17. Molecular Basis121 topic
  18. Molecular Docking121 topic
  19. Prion Diseases121 topic
  20. Virtual Screening121 topic

Where does Subashchandrabose Chinnathambi work?

Subashchandrabose Chinnathambi's main affiliation in the publication record is National Chemical Laboratory, India.

How many publications and citations does Subashchandrabose Chinnathambi have?

OpenAlex counts 176 works and 5,358 citations for Subashchandrabose Chinnathambi, an h-index of 46, ranking #23,532 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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