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Researcher · Tatarstan Academy of Sciences · Russia

T. M. Gainutdinov

T. M. Gainutdinov publishes mostly in Molecular Biology, Ecology and Aging, on topics such as Genetics, Aging, and Longevity in Model Organisms, Mitochondrial Function and Pathology and Physiological and biochemical adaptations.

ORCID 0000-0003-1723-1780 OpenAlex
World rank
#4,244,461
of 7,710,434 ranked researchers
Rank in Russia
#24,082
of 179,717
Works
31
Citations
317
Citations per work
10.2
h-index
6
i10-index 5Top 94% in Molecular Biology
T. M. Gainutdinov

What does T. M. Gainutdinov research?

T. M. Gainutdinov's work is mostly in Biochemistry, Genetics and Molecular Biology (29%), Environmental Science (10%), Neuroscience (6%).

Everything else: 14 works · 45.0% of their workLife Sciences: 11 works · 35.4% of their workBiochemistry, Genetics and Molecular Biology: 9 works · 29.3% of their workMolecular Biology: 6 works · 19.5% of their workAging: 3 works · 9.8% of their workNeuroscience: 2 works · 6.1% of their workCellular and Molecular Neuroscience: 2 works · 6.1% of their workPhysical Sciences: 5 works · 14.7% of their workEnvironmental Science: 3 works · 9.8% of their workEcology: 3 works · 9.8% of their workChemistry: 2 works · 4.9% of their workPhysical and Theoretical Chemistry: 2 works · 4.9% of their workHealth Sciences: 2 works · 4.9% of their workMedicine: 2 works · 4.9% of their workPhysiology: 2 works · 4.9% of their workTHEIR WORKT. M. Gainutdinov31works
Life Sciences35%Physical Sciences15%Health Sciences5%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
  1. Everything else1445.0%
    1. Biochemistry, Genetics and Molecular Biology929.3%
    2. Neuroscience26.1%
    1. Environmental Science39.8%
    2. Chemistry24.9%
    1. Medicine24.9%
  1. 1Molecular Biology20% of their works
  2. 2Ecology10% of their works
  3. 3Aging10% of their works
  4. 4Cellular and Molecular Neuroscience6% of their works
  5. 5Physiology5% of their works
  6. 6Physical and Theoretical Chemistry5% of their works

Research topics

Which keywords describe T. M. Gainutdinov's research?

The keywords of their largest research topics: Aging, Oxidative Stress, Mitochondrial Function, Genetic Regulation, Longevity, Climate Change, Metabolic Rate and Mitochondria.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 19 words, with their numbers
  1. Aging152 topics
  2. Oxidative Stress152 topics
  3. Mitochondrial Function122 topics
  4. Genetic Regulation81 topic
  5. Longevity81 topic
  6. Climate Change71 topic
  7. Metabolic Rate71 topic
  8. Mitochondria71 topic
  9. Reactive Oxygen Species71 topic
  10. Biochemical Genetics41 topic
  11. Isothermal Titration Calorimetry41 topic
  12. Metabolic Disorders41 topic
  13. Thermodynamics41 topic
  14. Microgravity31 topic
  15. Neurotransmission31 topic
  16. Spaceflight31 topic
  17. Synaptic Plasticity31 topic
  18. Light Harvesting21 topic
  19. Photosystem II21 topic

Where does T. M. Gainutdinov work?

T. M. Gainutdinov's main affiliation in the publication record is Tatarstan Academy of Sciences, Russia.

How many publications and citations does T. M. Gainutdinov have?

OpenAlex counts 31 works and 317 citations for T. M. Gainutdinov, an h-index of 6, ranking #4,244,461 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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