Takeshi Iwatsubo
Takeshi Iwatsubo publishes mostly in Physiology, Molecular Biology and Neurology, on topics such as Alzheimer's disease research and treatments, Dementia and Cognitive Impairment Research and Parkinson's Disease Mechanisms and Treatments.
- World rank
- #98,160 of 7,710,434 ranked researchers
- Rank in Japan
- #609 of 383,136
- Works
- 725
- Citations
- 76k
- Citations per work
- 104.5
What does Takeshi Iwatsubo research?
Takeshi Iwatsubo's work is mostly in Medicine (37%), Biochemistry, Genetics and Molecular Biology (16%), Neuroscience (10%).
Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.
The fields and subfields, with their numbers
- Health Sciences26836.9%
- Medicine26836.9%
- Everything else26736.8%
- Life Sciences19126.3%
- Biochemistry, Genetics and Molecular Biology11916.4%
- Neuroscience729.9%
- 1Physiology18% of their works
- 2Molecular Biology16% of their works
- 3Neurology10% of their works
- 4Psychiatry and Mental health8% of their works
- 5Cellular and Molecular Neuroscience6% of their works
- 6Neurology4% of their works
Research topics
- Alzheimer's disease research and treatments
- Dementia and Cognitive Impairment Research
- Parkinson's Disease Mechanisms and Treatments
- Cholinesterase and Neurodegenerative Diseases
- Cellular transport and secretion
- Nuclear Receptors and Signaling
- Prion Diseases and Protein Misfolding
- Neurological diseases and metabolism
Which keywords describe Takeshi Iwatsubo's research?
The keywords of their largest research topics: Neurodegeneration, Oxidative Stress, Amyloid Hypothesis, Tau Pathology, Alzheimer's Disease, Neuroimaging, Dopaminergic Neurons and Parkinson's Disease.
- Pantothenate Kinase
- Prion Diseases
- Nurr1
- Membrane Fusion
- Cholinesterase Inhibitors
- Neurodegenerative Diseases
- Genetic Risk Factors
- Dementia
- Parkinson's Disease
- Neuroimaging
- Tau Pathology
- Oxidative Stress
- Neurodegeneration
- Amyloid Hypothesis
- Alzheimer's Disease
- Dopaminergic Neurons
- Mitochondrial Dysfunction
- Mild Cognitive Impairment
- a-Synuclein
- Acetylcholinesterase
- Endocytosis
- NR4A2
- Molecular Basis
- Brain Iron Accumulation
Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.
All 24 words, with their numbers
- Neurodegeneration5114 topics
- Oxidative Stress4432 topics
- Amyloid Hypothesis3311 topic
- Tau Pathology3311 topic
- Alzheimer's Disease2032 topics
- Neuroimaging1732 topics
- Dopaminergic Neurons1492 topics
- Parkinson's Disease1492 topics
- Mitochondrial Dysfunction1432 topics
- Dementia1421 topic
- Mild Cognitive Impairment1421 topic
- Genetic Risk Factors1121 topic
- a-Synuclein1121 topic
- Neurodegenerative Diseases962 topics
- Acetylcholinesterase611 topic
- Cholinesterase Inhibitors611 topic
- Endocytosis401 topic
- Membrane Fusion401 topic
- NR4A2371 topic
- Nurr1371 topic
- Molecular Basis351 topic
- Prion Diseases351 topic
- Brain Iron Accumulation311 topic
- Pantothenate Kinase311 topic
Where does Takeshi Iwatsubo work?
Takeshi Iwatsubo's main affiliation in the publication record is The University of Tokyo, Japan.
How many publications and citations does Takeshi Iwatsubo have?
OpenAlex counts 725 works and 75,749 citations for Takeshi Iwatsubo, an h-index of 115, ranking #98,160 of 7,710,434 researchers worldwide on the quality score.
Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.
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