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Researcher · University of British Columbia · Canada

A. Jon Stoessl

A. Jon Stoessl publishes mostly in Neurology, Cellular and Molecular Neuroscience and Molecular Biology, on topics such as Parkinson's Disease Mechanisms and Treatments, Neurological disorders and treatments and Genetic Neurodegenerative Diseases.

ORCID 0000-0002-9778-7246 OpenAlex
World rank
#204,076
of 7,710,434 ranked researchers
Rank in Canada
#6,847
of 183,316
Works
450
Citations
32k
Citations per work
70.7
h-index
82
i10-index 288Top 0.6% in Neurology (Medicine)
A. Jon Stoessl

What does A. Jon Stoessl research?

A. Jon Stoessl's work is mostly in Medicine (37%), Neuroscience (22%), Biochemistry, Genetics and Molecular Biology (6%).

Health Sciences: 168 works · 37.4% of their workMedicine: 168 works · 37.4% of their workNeurology: 168 works · 37.4% of their workEverything else: 144 works · 32.1% of their workLife Sciences: 137 works · 30.5% of their workNeuroscience: 98 works · 21.7% of their workCellular and Molecular Neuroscience: 61 works · 13.5% of their workCognitive Neuroscience: 21 works · 4.6% of their workNeurology: 16 works · 3.6% of their workBiochemistry, Genetics and Molecular Biology: 27 works · 6.1% of their workMolecular Biology: 27 works · 6.1% of their workAgricultural and Biological Sciences: 12 works · 2.7% of their workPlant Science: 12 works · 2.7% of their workTHEIR WORKA. Jon Stoessl450works
Life Sciences30%Health Sciences37%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Medicine16837.4%
  2. Everything else14432.1%
    1. Neuroscience9821.7%
    2. Biochemistry, Genetics and Molecular Biology276.1%
    3. Agricultural and Biological Sciences122.7%
  1. 1Neurology37% of their works
  2. 2Cellular and Molecular Neuroscience14% of their works
  3. 3Molecular Biology6% of their works
  4. 4Cognitive Neuroscience5% of their works
  5. 5Neurology4% of their works
  6. 6Plant Science3% of their works

Research topics

Which keywords describe A. Jon Stoessl's research?

The keywords of their largest research topics: Parkinson's Disease, Mitochondrial Dysfunction, Neurological Disorders, Neurodegeneration, a-Synuclein, Dopaminergic Neurons, Deep Brain Stimulation and Subthalamic Nucleus.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 21 words, with their numbers
  1. Parkinson's Disease4122 topics
  2. Mitochondrial Dysfunction2823 topics
  3. Neurological Disorders2633 topics
  4. Neurodegeneration2322 topics
  5. Dopaminergic Neurons2091 topic
  6. a-Synuclein2091 topic
  7. Deep Brain Stimulation2031 topic
  8. Subthalamic Nucleus2031 topic
  9. Stress642 topics
  10. Huntington's Disease501 topic
  11. Protein Aggregation501 topic
  12. Dopamine421 topic
  13. Serotonin421 topic
  14. Botulinum Toxin331 topic
  15. Neurotoxins331 topic
  16. Neurotransmission271 topic
  17. Synaptic Plasticity271 topic
  18. Brain Iron Accumulation231 topic
  19. Pantothenate Kinase231 topic
  20. Neuropeptides221 topic
  21. Receptors221 topic

Where does A. Jon Stoessl work?

A. Jon Stoessl's main affiliation in the publication record is University of British Columbia, Canada.

How many publications and citations does A. Jon Stoessl have?

OpenAlex counts 450 works and 31,797 citations for A. Jon Stoessl, an h-index of 82, ranking #204,076 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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