Science Explorer Back to mapMap
Discussion
Researcher · Neurological Research Institute · United States

Aleksandar Bajić

Aleksandar Bajić publishes mostly in Molecular Biology, Cellular and Molecular Neuroscience and Genetics, on topics such as Genetics and Neurodevelopmental Disorders, Retinal Development and Disorders and Neuroscience and Neuropharmacology Research.

ORCID 0000-0002-1255-9835 OpenAlex
World rank
#1,370,767
of 7,710,434 ranked researchers
Rank in United States
#433,819
of 1,627,098
Works
44
Citations
1.4k
Citations per work
32.2
h-index
20
i10-index 25Top 33% in Molecular Biology
Aleksandar Bajić

What does Aleksandar Bajić research?

Aleksandar Bajić's work is mostly in Biochemistry, Genetics and Molecular Biology (37%), Neuroscience (19%), Medicine (5%).

Life Sciences: 24 works · 55.6% of their workBiochemistry, Genetics and Molecular Biology: 16 works · 36.5% of their workMolecular Biology: 13 works · 30.2% of their workGenetics: 3 works · 6.3% of their workNeuroscience: 8 works · 19.1% of their workCellular and Molecular Neuroscience: 5 works · 11.1% of their workDevelopmental Neuroscience: 2 works · 4.0% of their workNeurology: 2 works · 4.0% of their workEverything else: 17 works · 39.6% of their workHealth Sciences: 2 works · 4.8% of their workMedicine: 2 works · 4.8% of their workPhysiology: 2 works · 4.8% of their workTHEIR WORKAleksandar Bajić44works
Life Sciences56%Health Sciences5%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology1636.5%
    2. Neuroscience819.1%
  2. Everything else1739.6%
    1. Medicine24.8%
  1. 1Molecular Biology30% of their works
  2. 2Cellular and Molecular Neuroscience11% of their works
  3. 3Genetics6% of their works
  4. 4Physiology5% of their works
  5. 5Neurology4% of their works
  6. 6Developmental Neuroscience4% of their works

Research topics

Which keywords describe Aleksandar Bajić's research?

The keywords of their largest research topics: Dendritic Spines, Brain Development, Fragile X Syndrome, Gene Therapy, Retinitis Pigmentosa, Rett Syndrome, Neurotransmission and Synaptic Plasticity.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 18 words, with their numbers
  1. Dendritic Spines112 topics
  2. Brain Development102 topics
  3. Fragile X Syndrome61 topic
  4. Gene Therapy61 topic
  5. Retinitis Pigmentosa61 topic
  6. Rett Syndrome61 topic
  7. Neurotransmission51 topic
  8. Synaptic Plasticity51 topic
  9. Adult Stem Cells41 topic
  10. Channelrhodopsin41 topic
  11. Endocytosis41 topic
  12. Membrane Fusion41 topic
  13. Neurogenesis41 topic
  14. Optogenetics41 topic
  15. DNA Methylation31 topic
  16. Histone Modification31 topic
  17. Mitochondria31 topic
  18. Reactive Oxygen Species31 topic

Where does Aleksandar Bajić work?

Aleksandar Bajić's main affiliation in the publication record is Neurological Research Institute, United States.

How many publications and citations does Aleksandar Bajić have?

OpenAlex counts 44 works and 1,418 citations for Aleksandar Bajić, an h-index of 20, ranking #1,370,767 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

Ask a question or share a note about Aleksandar Bajić. Keep it to the research: no personal attacks, advertising or links. Any reader can report a post, which collapses it for everyone until it is opened.