Science Explorer Back to mapMap
Discussion
Researcher · Semmelweis University · Hungary

Attila Mócsai

Attila Mócsai publishes mostly in Immunology, Molecular Biology and Immunology and Allergy, on topics such as Cell Adhesion Molecules Research, Neutrophil, Myeloperoxidase and Oxidative Mechanisms and Immune Response and Inflammation.

ORCID 0000-0002-0512-1157 OpenAlex
World rank
#389,945
of 7,710,434 ranked researchers
Rank in Hungary
#357
of 25,218
Works
177
Citations
13k
Citations per work
72.7
h-index
51
i10-index 93Top 4% in Immunology
Attila Mócsai

What does Attila Mócsai research?

Attila Mócsai's work is mostly in Immunology and Microbiology (28%), Medicine (25%), Biochemistry, Genetics and Molecular Biology (14%).

Life Sciences: 73 works · 41.1% of their workImmunology and Microbiology: 49 works · 27.5% of their workImmunology: 49 works · 27.5% of their workBiochemistry, Genetics and Molecular Biology: 24 works · 13.6% of their workMolecular Biology: 24 works · 13.6% of their workEverything else: 59 works · 33.6% of their workHealth Sciences: 45 works · 25.3% of their workMedicine: 45 works · 25.3% of their workImmunology and Allergy: 20 works · 11.3% of their workRheumatology: 9 works · 5.3% of their workOncology: 9 works · 5.1% of their workPhysiology: 6 works · 3.6% of their workTHEIR WORKAttila Mócsai177works
Life Sciences41%Health Sciences25%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Immunology and Microbiology4927.5%
    2. Biochemistry, Genetics and Molecular Biology2413.6%
  2. Everything else5933.6%
    1. Medicine4525.3%
  1. 1Immunology28% of their works
  2. 2Molecular Biology14% of their works
  3. 3Immunology and Allergy11% of their works
  4. 4Rheumatology5% of their works
  5. 5Oncology5% of their works
  6. 6Physiology4% of their works

Research topics

Which keywords describe Attila Mócsai's research?

The keywords of their largest research topics: Inflammation, Cell Adhesion, Integrins, Neutrophil Extracellular Traps, Reactive Oxygen Species, Pattern Recognition Receptors, Toll-like Receptors and Autoantibodies.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 19 words, with their numbers
  1. Inflammation1454 topics
  2. Cell Adhesion521 topic
  3. Integrins521 topic
  4. Neutrophil Extracellular Traps441 topic
  5. Reactive Oxygen Species441 topic
  6. Pattern Recognition Receptors291 topic
  7. Toll-like Receptors291 topic
  8. Autoantibodies212 topics
  9. Diagnosis212 topics
  10. Allergic Disease201 topic
  11. Mast Cells201 topic
  12. Bone Resorption121 topic
  13. Fc Receptors121 topic
  14. Osteoclast Differentiation121 topic
  15. Therapeutic Antibodies121 topic
  16. Pemphigoid111 topic
  17. Rituximab111 topic
  18. Chronic Spontaneous Urticaria101 topic
  19. Urticaria101 topic

Where does Attila Mócsai work?

Attila Mócsai's main affiliation in the publication record is Semmelweis University, Hungary.

How many publications and citations does Attila Mócsai have?

OpenAlex counts 177 works and 12,876 citations for Attila Mócsai, an h-index of 51, ranking #389,945 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

Ask a question or share a note about Attila Mócsai. Keep it to the research: no personal attacks, advertising or links. Any reader can report a post, which collapses it for everyone until it is opened.