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Researcher · NeuroSearch (Denmark) · Denmark

Daniel Brunicardi Timmermann

Daniel Brunicardi Timmermann publishes mostly in Molecular Biology, Law and Cellular and Molecular Neuroscience, on topics such as Nicotinic Acetylcholine Receptors Study, Receptor Mechanisms and Signaling and Ion channel regulation and function.

OpenAlex
World rank
#3,044,094
of 7,710,434 ranked researchers
Rank in Denmark
#22,433
of 43,819
Works
37
Citations
1.6k
Citations per work
42.1
h-index
19
i10-index 23Top 36% in Molecular Biology
Daniel Brunicardi Timmermann

What does Daniel Brunicardi Timmermann research?

Daniel Brunicardi Timmermann's work is mostly in Biochemistry, Genetics and Molecular Biology (40%), Social Sciences (18%), Neuroscience (13%).

Life Sciences: 19 works · 52.5% of their workBiochemistry, Genetics and Molecular Biology: 15 works · 39.6% of their workMolecular Biology: 15 works · 39.6% of their workNeuroscience: 5 works · 12.9% of their workCellular and Molecular Neuroscience: 4 works · 9.9% of their workNeurology: 1 works · 3.0% of their workEverything else: 9 works · 23.7% of their workSocial Sciences: 7 works · 17.9% of their workSocial Sciences: 7 works · 17.9% of their workLaw: 6 works · 14.9% of their workSociology and Political Science: 1 works · 3.0% of their workHealth Sciences: 2 works · 5.9% of their workMedicine: 2 works · 5.9% of their workPublic Health, Environmental and Occupational Health: 2 works · 5.9% of their workTHEIR WORKDaniel BrunicardiTimmermann37works
Life Sciences52%Health Sciences6%Social Sciences18%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology1539.6%
    2. Neuroscience512.9%
  2. Everything else923.7%
    1. Social Sciences717.9%
    1. Medicine25.9%
  1. 1Molecular Biology40% of their works
  2. 2Law15% of their works
  3. 3Cellular and Molecular Neuroscience10% of their works
  4. 4Public Health, Environmental and Occupational Health6% of their works
  5. 5Sociology and Political Science3% of their works
  6. 6Neurology3% of their works

Research topics

Which keywords describe Daniel Brunicardi Timmermann's research?

The keywords of their largest research topics: Crystal Structure, Cell Signaling, Neuronal Signaling, Nicotinic Acetylcholine Receptors, G-Protein-Coupled Receptors, Signal Transduction, Ion Channels and Voltage-Gated Channels.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most.

All 18 words, with their numbers
  1. Crystal Structure292 topics
  2. Cell Signaling212 topics
  3. Neuronal Signaling161 topic
  4. Nicotinic Acetylcholine Receptors161 topic
  5. G-Protein-Coupled Receptors131 topic
  6. Signal Transduction131 topic
  7. Ion Channels81 topic
  8. Voltage-Gated Channels81 topic
  9. Legal Methodology71 topic
  10. Neurotransmission71 topic
  11. Party Competition71 topic
  12. Synaptic Plasticity71 topic
  13. Artificial Intelligence61 topic
  14. Data Protection61 topic
  15. Medical Ethics61 topic
  16. Patient Rights61 topic
  17. Cholinergic Antiinflammatory Pathway31 topic
  18. Vagus Nerve Stimulation31 topic

Where does Daniel Brunicardi Timmermann work?

Daniel Brunicardi Timmermann's main affiliation in the publication record is NeuroSearch (Denmark), Denmark.

How many publications and citations does Daniel Brunicardi Timmermann have?

OpenAlex counts 37 works and 1,557 citations for Daniel Brunicardi Timmermann, an h-index of 19, ranking #3,044,094 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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