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Researcher · deCODE Genetics (Iceland) · Iceland

Daniel Fannar Gudbjartsson

Daniel Fannar Gudbjartsson publishes mostly in Genetics, Molecular Biology and Cardiology and Cardiovascular Medicine, on topics such as Genetic Associations and Epidemiology, Genomics and Rare Diseases and Epigenetics and DNA Methylation.

ORCID 0000-0002-5222-9857 OpenAlex
World rank
#303
of 7,710,434 ranked researchers
Rank in Iceland
#4
of 2,050
Works
524
Citations
88k
Citations per work
167.8
h-index
143
i10-index 349Top 0.1% in Genetics (Biochemistry, Genetics and Molecular Biology)
Daniel Fannar Gudbjartsson

What does Daniel Fannar Gudbjartsson research?

Daniel Fannar Gudbjartsson's work is mostly in Biochemistry, Genetics and Molecular Biology (45%), Medicine (13%), Immunology and Microbiology (3%).

Life Sciences: 248 works · 47.3% of their workBiochemistry, Genetics and Molecular Biology: 234 works · 44.6% of their workGenetics: 137 works · 26.1% of their workMolecular Biology: 97 works · 18.5% of their workImmunology and Microbiology: 14 works · 2.7% of their workImmunology: 14 works · 2.7% of their workEverything else: 207 works · 39.5% of their workHealth Sciences: 69 works · 13.2% of their workMedicine: 69 works · 13.2% of their workCardiology and Cardiovascular Medicine: 36 works · 6.9% of their workSurgery: 17 works · 3.3% of their workEndocrinology, Diabetes and Metabolism: 16 works · 3.0% of their workTHEIR WORKDaniel FannarGudbjartsson524works
Life Sciences47%Health Sciences13%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology23444.6%
    2. Immunology and Microbiology142.7%
  2. Everything else20739.5%
    1. Medicine6913.2%
  1. 1Genetics26% of their works
  2. 2Molecular Biology18% of their works
  3. 3Cardiology and Cardiovascular Medicine7% of their works
  4. 4Surgery3% of their works
  5. 5Endocrinology, Diabetes and Metabolism3% of their works
  6. 6Immunology3% of their works

Research topics

Which keywords describe Daniel Fannar Gudbjartsson's research?

The keywords of their largest research topics: Genetic Variation, Genome-Wide Association, Haplotype Mapping, Genetic Variants, Sequence Interpretation, DNA Methylation, Histone Modification and Copy Number Variation.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 17 words, with their numbers
  1. Genetic Variation1752 topics
  2. Genome-Wide Association1561 topic
  3. Haplotype Mapping1561 topic
  4. Genetic Variants461 topic
  5. Sequence Interpretation461 topic
  6. DNA Methylation321 topic
  7. Histone Modification321 topic
  8. Copy Number Variation271 topic
  9. Genomic Rearrangements271 topic
  10. RNA methylation241 topic
  11. m6A-seq241 topic
  12. Cardiomyocyte Renewal221 topic
  13. Heart Regeneration221 topic
  14. Alternative Splicing191 topic
  15. Genome-wide Association Studies191 topic
  16. Quantitative Trait Loci191 topic
  17. RNA-Binding Proteins191 topic

Where does Daniel Fannar Gudbjartsson work?

Daniel Fannar Gudbjartsson's main affiliation in the publication record is deCODE Genetics (Iceland), Iceland.

How many publications and citations does Daniel Fannar Gudbjartsson have?

OpenAlex counts 524 works and 87,949 citations for Daniel Fannar Gudbjartsson, an h-index of 143, ranking #303 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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