Douglas Kazutoshi Sato
Douglas Kazutoshi Sato publishes mostly in Pathology and Forensic Medicine, Neurology and Rheumatology, on topics such as Multiple Sclerosis Research Studies, Peripheral Neuropathies and Disorders and Autoimmune Neurological Disorders and Treatments.
- World rank
- #374,010 of 7,710,434 ranked researchers
- Rank in Brazil
- #1,643 of 253,246
- Works
- 195
- Citations
- 9.5k
- Citations per work
- 48.7
What does Douglas Kazutoshi Sato research?
Douglas Kazutoshi Sato's work is mostly in Medicine (62%), Immunology and Microbiology (4%), Biochemistry, Genetics and Molecular Biology (3%).
Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.
The fields and subfields, with their numbers
- Health Sciences12162.0%
- Medicine12162.0%
- Everything else6131.4%
- Life Sciences136.6%
- 1Pathology and Forensic Medicine27% of their works
- 2Neurology27% of their works
- 3Rheumatology6% of their works
- 4Immunology4% of their works
- 5Molecular Biology3% of their works
- 6Epidemiology3% of their works
Research topics
- Multiple Sclerosis Research Studies
- Peripheral Neuropathies and Disorders
- Autoimmune Neurological Disorders and Treatments
- Systemic Lupus Erythematosus Research
- Systemic Sclerosis and Related Diseases
- Long-Term Effects of COVID-19
- Neurogenesis and neuroplasticity mechanisms
- Neuroinflammation and Neurodegeneration Mechanisms
Which keywords describe Douglas Kazutoshi Sato's research?
The keywords of their largest research topics: Cognitive Impairment, Diagnostic Criteria, Multiple Sclerosis, Neuromyelitis Optica, Neurological Disorders, Treatment, Chronic Inflammatory Demyelinating Polyradiculoneuropathy and Guillain-Barré Syndrome.
- Neuroinflammation
- Microglia
- Post-acute COVID-19 Syndrome
- Systemic Sclerosis
- Brain Development
- Antiphospholipid Syndrome
- Anti-NMDA Receptor Encephalitis
- Guillain-Barré Syndrome
- Treatment
- Neuromyelitis Optica
- Diagnostic Criteria
- Cognitive Impairment
- Multiple Sclerosis
- Neurological Disorders
- Chronic Inflammatory Demyelinating Polyradiculoneuropathy
- Autoantibodies
- Paraneoplastic Syndromes
- Systemic Lupus Erythematosus
- Fibrosis
- Neurologic Manifestations
- Adult Stem Cells
- Neurogenesis
Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.
All 22 words, with their numbers
- Cognitive Impairment1693 topics
- Diagnostic Criteria1602 topics
- Multiple Sclerosis1231 topic
- Neuromyelitis Optica1231 topic
- Neurological Disorders1212 topics
- Treatment982 topics
- Chronic Inflammatory Demyelinating Polyradiculoneuropathy841 topic
- Guillain-Barré Syndrome841 topic
- Autoantibodies753 topics
- Anti-NMDA Receptor Encephalitis371 topic
- Paraneoplastic Syndromes371 topic
- Antiphospholipid Syndrome241 topic
- Systemic Lupus Erythematosus241 topic
- Brain Development142 topics
- Fibrosis141 topic
- Systemic Sclerosis141 topic
- Neurologic Manifestations91 topic
- Post-acute COVID-19 Syndrome91 topic
- Adult Stem Cells71 topic
- Microglia71 topic
- Neurogenesis71 topic
- Neuroinflammation71 topic
Where does Douglas Kazutoshi Sato work?
Douglas Kazutoshi Sato's main affiliation in the publication record is Pontifícia Universidade Católica do Rio Grande do Sul, Brazil.
How many publications and citations does Douglas Kazutoshi Sato have?
OpenAlex counts 195 works and 9,503 citations for Douglas Kazutoshi Sato, an h-index of 42, ranking #374,010 of 7,710,434 researchers worldwide on the quality score.
Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.
Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.
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