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Researcher · University of Groningen · Netherlands

Ellen A. A. Nollen

Ellen A. A. Nollen publishes mostly in Molecular Biology, Aging and Physiology, on topics such as Genetics, Aging, and Longevity in Model Organisms, Alzheimer's disease research and treatments and Mitochondrial Function and Pathology.

ORCID 0000-0003-3740-6373 OpenAlex
World rank
#16,665
of 7,710,434 ranked researchers
Rank in Netherlands
#271
of 107,149
Works
5,628
Citations
8.1k
Citations per work
1.4
h-index
37
i10-index 55Top 8.9% in Molecular Biology
Ellen A. A. Nollen

What does Ellen A. A. Nollen research?

Ellen A. A. Nollen's work is mostly in Biochemistry, Genetics and Molecular Biology (43%), Medicine (18%), Neuroscience (6%).

Life Sciences: 2,803 works · 49.8% of their workBiochemistry, Genetics and Molecular Biology: 2,437 works · 43.3% of their workMolecular Biology: 1,159 works · 20.6% of their workAging: 957 works · 17.0% of their workCell Biology: 321 works · 5.7% of their workNeuroscience: 366 works · 6.5% of their workCellular and Molecular Neuroscience: 366 works · 6.5% of their workEverything else: 1,823 works · 32.4% of their workHealth Sciences: 1,002 works · 17.8% of their workMedicine: 1,002 works · 17.8% of their workPhysiology: 591 works · 10.5% of their workNeurology: 411 works · 7.3% of their workTHEIR WORKEllen A. A.Nollen5,628works
Life Sciences50%Health Sciences18%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
  1. Life Sciences2,80349.8%
    1. Biochemistry, Genetics and Molecular Biology2,43743.3%
    2. Neuroscience3666.5%
  2. Everything else1,82332.4%
  3. Health Sciences1,00217.8%
    1. Medicine1,00217.8%
  1. 1Molecular Biology21% of their works
  2. 2Aging17% of their works
  3. 3Physiology10% of their works
  4. 4Neurology7% of their works
  5. 5Cellular and Molecular Neuroscience6% of their works
  6. 6Cell Biology6% of their works

Research topics

Which keywords describe Ellen A. A. Nollen's research?

The keywords of their largest research topics: Oxidative Stress, Aging, Proteostasis, Genetic Regulation, Longevity, Mitochondrial Dysfunction, Neurodegeneration and Neuroinflammation.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 25 words, with their numbers
  1. Oxidative Stress1066 topics
  2. Aging552 topics
  3. Proteostasis542 topics
  4. Genetic Regulation421 topic
  5. Longevity421 topic
  6. Mitochondrial Dysfunction353 topics
  7. Neurodegeneration312 topics
  8. Neuroinflammation272 topics
  9. Cell Death252 topics
  10. Protein Folding242 topics
  11. Chaperones222 topics
  12. Amyloid Hypothesis191 topic
  13. Tau Pathology191 topic
  14. Mitochondria131 topic
  15. Reactive Oxygen Species131 topic
  16. Endoplasmic Reticulum121 topic
  17. Heat Shock Proteins121 topic
  18. Molecular Chaperones121 topic
  19. Parkinson's Disease121 topic
  20. Stress121 topic
  21. a-Synuclein121 topic
  22. Huntington's Disease101 topic
  23. Protein Aggregation101 topic
  24. Cytokines81 topic
  25. Depression81 topic

Where does Ellen A. A. Nollen work?

Ellen A. A. Nollen's main affiliation in the publication record is University of Groningen, Netherlands.

How many publications and citations does Ellen A. A. Nollen have?

OpenAlex counts 5,628 works and 8,134 citations for Ellen A. A. Nollen, an h-index of 37, ranking #16,665 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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