Science Explorer Back to mapMap
Discussion
Researcher · University of Hassan II Casablanca · Morocco

Fatima Ailal

Fatima Ailal publishes mostly in Immunology, Genetics and Infectious Diseases, on topics such as Immunodeficiency and Autoimmune Disorders, Blood disorders and treatments and Immune Cell Function and Interaction.

ORCID 0000-0001-5824-3193 OpenAlex
World rank
#1,559,125
of 7,710,434 ranked researchers
Rank in Morocco
#1,198
of 14,526
Works
145
Citations
4.3k
Citations per work
29.9
h-index
24
i10-index 37Top 23% in Immunology
Fatima Ailal

What does Fatima Ailal research?

Fatima Ailal's work is mostly in Immunology and Microbiology (38%), Medicine (24%), Biochemistry, Genetics and Molecular Biology (10%).

Life Sciences: 70 works · 48.2% of their workImmunology and Microbiology: 55 works · 37.9% of their workImmunology: 55 works · 37.9% of their workBiochemistry, Genetics and Molecular Biology: 15 works · 10.3% of their workGenetics: 15 works · 10.3% of their workEverything else: 40 works · 27.9% of their workHealth Sciences: 35 works · 23.9% of their workMedicine: 35 works · 23.9% of their workInfectious Diseases: 12 works · 8.1% of their workEpidemiology: 11 works · 7.3% of their workSurgery: 7 works · 4.6% of their workPulmonary and Respiratory Medicine: 6 works · 3.9% of their workTHEIR WORKFatima Ailal145works
Life Sciences48%Health Sciences24%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Immunology and Microbiology5537.9%
    2. Biochemistry, Genetics and Molecular Biology1510.3%
  2. Everything else4027.9%
    1. Medicine3523.9%
  1. 1Immunology38% of their works
  2. 2Genetics10% of their works
  3. 3Infectious Diseases8% of their works
  4. 4Epidemiology7% of their works
  5. 5Surgery5% of their works
  6. 6Pulmonary and Respiratory Medicine4% of their works

Research topics

Which keywords describe Fatima Ailal's research?

The keywords of their largest research topics: Genetic Mutations, Immune Dysregulation, Primary Immunodeficiency, Congenital Disorders, Neutropenia, Autoimmunity, Diagnosis and Epidemiology.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 20 words, with their numbers
  1. Genetic Mutations1222 topics
  2. Immune Dysregulation891 topic
  3. Primary Immunodeficiency891 topic
  4. Congenital Disorders331 topic
  5. Neutropenia331 topic
  6. Autoimmunity222 topics
  7. Diagnosis202 topics
  8. Epidemiology202 topics
  9. NK Cell Recognition201 topic
  10. Natural Killer Cells201 topic
  11. Foxp3151 topic
  12. TGF-ß151 topic
  13. Mycobacterium tuberculosis121 topic
  14. Tuberculosis121 topic
  15. Mycobacterium avium81 topic
  16. Nontuberculous Mycobacteria81 topic
  17. Innate Memory71 topic
  18. Neutrophil Extracellular Traps71 topic
  19. Reactive Oxygen Species71 topic
  20. Trained Immunity71 topic

Where does Fatima Ailal work?

Fatima Ailal's main affiliation in the publication record is University of Hassan II Casablanca, Morocco.

How many publications and citations does Fatima Ailal have?

OpenAlex counts 145 works and 4,331 citations for Fatima Ailal, an h-index of 24, ranking #1,559,125 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

Ask a question or share a note about Fatima Ailal. Keep it to the research: no personal attacks, advertising or links. Any reader can report a post, which collapses it for everyone until it is opened.