J. Christopher Corton
J. Christopher Corton publishes mostly in Molecular Biology, Health, Toxicology and Mutagenesis and Cancer Research, on topics such as Peroxisome Proliferator-Activated Receptors, Effects and risks of endocrine disrupting chemicals and Carcinogens and Genotoxicity Assessment.
- World rank
- #423,452 of 7,710,434 ranked researchers
- Rank in United States
- #149,495 of 1,627,098
- Works
- 160
- Citations
- 14k
- Citations per work
- 88.9
What does J. Christopher Corton research?
J. Christopher Corton's work is mostly in Biochemistry, Genetics and Molecular Biology (45%), Environmental Science (12%), Medicine (6%).
Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.
The fields and subfields, with their numbers
- Life Sciences7848.6%
- Everything else4729.2%
- Physical Sciences2616.2%
- Environmental Science1912.0%
- Computer Science74.2%
- Health Sciences106.0%
- Medicine106.0%
- 1Molecular Biology35% of their works
- 2Health, Toxicology and Mutagenesis12% of their works
- 3Cancer Research10% of their works
- 4Oncology6% of their works
- 5Computational Theory and Mathematics4% of their works
- 6Pharmacology4% of their works
Research topics
- Peroxisome Proliferator-Activated Receptors
- Effects and risks of endocrine disrupting chemicals
- Carcinogens and Genotoxicity Assessment
- Drug Transport and Resistance Mechanisms
- Gene expression and cancer classification
- Computational Drug Discovery Methods
- Molecular Biology Techniques and Applications
- Per- and polyfluoroalkyl substances research
Which keywords describe J. Christopher Corton's research?
The keywords of their largest research topics: Nuclear Receptors, Health Effects, Peroxisome Proliferator-Activated Receptors, PPARs, Machine Learning, Data Analysis, Gene Expression and Bisphenol A.
- Perfluoroalkyl Substances
- Quantitative
- Molecular Docking
- Differential Expression
- ATP-dependent Transporters
- Comet Assay
- Bisphenol A
- Data Analysis
- Peroxisome Proliferator-Activated Receptors
- Health Effects
- Nuclear Receptors
- PPARs
- Machine Learning
- Gene Expression
- Endocrine-Disrupting Chemicals
- Genotoxicity Testing
- Multidrug Resistance
- Microarray
- Virtual Screening
- Real-Time PCR
- Polyfluoroalkyl Compounds
Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.
All 21 words, with their numbers
- Nuclear Receptors682 topics
- Health Effects492 topics
- PPARs441 topic
- Peroxisome Proliferator-Activated Receptors441 topic
- Machine Learning382 topics
- Data Analysis352 topics
- Gene Expression352 topics
- Bisphenol A341 topic
- Endocrine-Disrupting Chemicals341 topic
- Comet Assay261 topic
- Genotoxicity Testing261 topic
- ATP-dependent Transporters241 topic
- Multidrug Resistance241 topic
- Differential Expression191 topic
- Microarray191 topic
- Molecular Docking191 topic
- Virtual Screening191 topic
- Quantitative161 topic
- Real-Time PCR161 topic
- Perfluoroalkyl Substances151 topic
- Polyfluoroalkyl Compounds151 topic
Where does J. Christopher Corton work?
J. Christopher Corton's main affiliation in the publication record is Research Triangle Park Foundation, United States.
How many publications and citations does J. Christopher Corton have?
OpenAlex counts 160 works and 14,221 citations for J. Christopher Corton, an h-index of 50, ranking #423,452 of 7,710,434 researchers worldwide on the quality score.
Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.
Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.
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