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Researcher · St Olav's University Hospital · Norway

Jan Olav Aasly

Jan Olav Aasly publishes mostly in Neurology, Molecular Biology and Physiology, on topics such as Parkinson's Disease Mechanisms and Treatments, Neurological disorders and treatments and Neurological diseases and metabolism.

ORCID 0000-0002-0558-7686 OpenAlex
World rank
#257,972
of 7,710,434 ranked researchers
Rank in Norway
#1,490
of 33,005
Works
310
Citations
22k
Citations per work
70.2
h-index
68
i10-index 211Top 1.2% in Neurology (Medicine)
Jan Olav Aasly

What does Jan Olav Aasly research?

Jan Olav Aasly's work is mostly in Medicine (39%), Biochemistry, Genetics and Molecular Biology (15%), Neuroscience (15%).

Health Sciences: 121 works · 39.0% of their workMedicine: 121 works · 39.0% of their workNeurology: 95 works · 30.7% of their workPhysiology: 26 works · 8.3% of their workEverything else: 98 works · 31.7% of their workLife Sciences: 91 works · 29.3% of their workBiochemistry, Genetics and Molecular Biology: 46 works · 14.7% of their workMolecular Biology: 33 works · 10.8% of their workGenetics: 12 works · 3.9% of their workNeuroscience: 45 works · 14.6% of their workCellular and Molecular Neuroscience: 25 works · 8.1% of their workNeurology: 20 works · 6.5% of their workTHEIR WORKJan Olav Aasly310works
Life Sciences29%Health Sciences39%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Medicine12139.0%
  2. Everything else9831.7%
    1. Biochemistry, Genetics and Molecular Biology4614.7%
    2. Neuroscience4514.6%
  1. 1Neurology31% of their works
  2. 2Molecular Biology11% of their works
  3. 3Physiology8% of their works
  4. 4Cellular and Molecular Neuroscience8% of their works
  5. 5Neurology6% of their works
  6. 6Genetics4% of their works

Research topics

Which keywords describe Jan Olav Aasly's research?

The keywords of their largest research topics: Parkinson's Disease, Neurodegeneration, Mitochondrial Dysfunction, Oxidative Stress, Dopaminergic Neurons, a-Synuclein, Genetic Risk Factors and Deep Brain Stimulation.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 23 words, with their numbers
  1. Parkinson's Disease2895 topics
  2. Neurodegeneration2604 topics
  3. Mitochondrial Dysfunction2303 topics
  4. Oxidative Stress1912 topics
  5. Dopaminergic Neurons1862 topics
  6. Genetic Risk Factors1631 topic
  7. a-Synuclein1631 topic
  8. Deep Brain Stimulation722 topics
  9. Neurological Disorders722 topics
  10. Basal Ganglia541 topic
  11. Subthalamic Nucleus541 topic
  12. Brain Iron Accumulation461 topic
  13. Pantothenate Kinase461 topic
  14. Enzyme Replacement Therapy311 topic
  15. Lysosomal Storage Disorders311 topic
  16. Amyloid Hypothesis281 topic
  17. Tau Pathology281 topic
  18. NR4A2231 topic
  19. Nurr1231 topic
  20. Huntington's Disease211 topic
  21. Protein Aggregation211 topic
  22. Camptocormia181 topic
  23. Dropped Head Syndrome181 topic

Where does Jan Olav Aasly work?

Jan Olav Aasly's main affiliation in the publication record is St Olav's University Hospital, Norway.

How many publications and citations does Jan Olav Aasly have?

OpenAlex counts 310 works and 21,760 citations for Jan Olav Aasly, an h-index of 68, ranking #257,972 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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