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Researcher · Simula Research Laboratory · Norway

Johan E. Hake

Johan E. Hake publishes mostly in Molecular Biology, Cardiology and Cardiovascular Medicine and Cellular and Molecular Neuroscience, on topics such as Cardiac electrophysiology and arrhythmias, Ion channel regulation and function and Neuroscience and Neural Engineering.

ORCID 0000-0002-4042-0128 OpenAlex
World rank
#3,663,292
of 7,710,434 ranked researchers
Rank in Norway
#21,508
of 33,005
Works
41
Citations
2.7k
Citations per work
66.8
h-index
15
i10-index 17Top 50% in Molecular Biology
Johan E. Hake

What does Johan E. Hake research?

Johan E. Hake's work is mostly in Biochemistry, Genetics and Molecular Biology (31%), Medicine (26%), Neuroscience (14%).

Life Sciences: 18 works · 45.0% of their workBiochemistry, Genetics and Molecular Biology: 13 works · 31.3% of their workMolecular Biology: 12 works · 28.4% of their workBiophysics: 1 works · 2.9% of their workNeuroscience: 6 works · 13.7% of their workCellular and Molecular Neuroscience: 6 works · 13.7% of their workHealth Sciences: 10 works · 25.5% of their workMedicine: 10 works · 25.5% of their workCardiology and Cardiovascular Medicine: 10 works · 25.5% of their workEverything else: 10 works · 23.7% of their workPhysical Sciences: 2 works · 5.8% of their workComputer Science: 1 works · 2.9% of their workArtificial Intelligence: 1 works · 2.9% of their workEngineering: 1 works · 2.9% of their workElectrical and Electronic Engineering: 1 works · 2.9% of their workTHEIR WORKJohan E. Hake41works
Life Sciences45%Physical Sciences6%Health Sciences26%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology1331.3%
    2. Neuroscience613.7%
    1. Medicine1025.5%
  3. Everything else1023.7%
    1. Computer Science12.9%
    2. Engineering12.9%
  1. 1Molecular Biology28% of their works
  2. 2Cardiology and Cardiovascular Medicine26% of their works
  3. 3Cellular and Molecular Neuroscience14% of their works
  4. 4Artificial Intelligence3% of their works
  5. 5Biophysics3% of their works
  6. 6Electrical and Electronic Engineering3% of their works

Research topics

Which keywords describe Johan E. Hake's research?

The keywords of their largest research topics: Ion Channels, Arrhythmias, Long-QT Syndrome, Cell Signaling, Calcium Channels, Voltage-Gated Channels, Electrode Arrays and Neural Stimulation.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 18 words, with their numbers
  1. Ion Channels382 topics
  2. Arrhythmias221 topic
  3. Long-QT Syndrome221 topic
  4. Cell Signaling212 topics
  5. Calcium Channels161 topic
  6. Voltage-Gated Channels161 topic
  7. Electrode Arrays101 topic
  8. Neural Stimulation101 topic
  9. Crystal Structure51 topic
  10. G-Protein-Coupled Receptors51 topic
  11. Cardiomyopathies31 topic
  12. Genetic Mutations31 topic
  13. Neurotransmission31 topic
  14. Ribosome31 topic
  15. Synaptic Plasticity31 topic
  16. Translation31 topic
  17. Breeding21 topic
  18. Genomics21 topic

Where does Johan E. Hake work?

Johan E. Hake's main affiliation in the publication record is Simula Research Laboratory, Norway.

How many publications and citations does Johan E. Hake have?

OpenAlex counts 41 works and 2,737 citations for Johan E. Hake, an h-index of 15, ranking #3,663,292 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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