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Researcher · Tulane University · United States

John A. McLachlan

John A. McLachlan publishes mostly in Health, Toxicology and Mutagenesis, Genetics and Molecular Biology, on topics such as Effects and risks of endocrine disrupting chemicals, Estrogen and related hormone effects and Reproductive System and Pregnancy.

ORCID 0000-0002-9039-1361 OpenAlex
World rank
#92,430
of 7,710,434 ranked researchers
Rank in United States
#35,577
of 1,627,098
Works
296
Citations
22k
Citations per work
74.2
h-index
78
i10-index 226Top 0.7% in Health, Toxicology and Mutagenesis
John A. McLachlan

What does John A. McLachlan research?

John A. McLachlan's work is mostly in Biochemistry, Genetics and Molecular Biology (31%), Environmental Science (21%), Immunology and Microbiology (3%).

Everything else: 123 works · 41.4% of their workLife Sciences: 102 works · 34.3% of their workBiochemistry, Genetics and Molecular Biology: 92 works · 31.0% of their workGenetics: 46 works · 15.6% of their workMolecular Biology: 36 works · 12.2% of their workCancer Research: 9 works · 3.2% of their workImmunology and Microbiology: 10 works · 3.3% of their workImmunology: 10 works · 3.3% of their workPhysical Sciences: 63 works · 21.3% of their workEnvironmental Science: 63 works · 21.3% of their workHealth, Toxicology and Mutagenesis: 63 works · 21.3% of their workHealth Sciences: 9 works · 3.0% of their workMedicine: 9 works · 3.0% of their workPathology and Forensic Medicine: 9 works · 3.0% of their workTHEIR WORKJohn A. McLachlan296works
Life Sciences34%Physical Sciences21%Health Sciences3%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
  1. Everything else12341.4%
    1. Biochemistry, Genetics and Molecular Biology9231.0%
    2. Immunology and Microbiology103.3%
    1. Environmental Science6321.3%
    1. Medicine93.0%
  1. 1Health, Toxicology and Mutagenesis21% of their works
  2. 2Genetics16% of their works
  3. 3Molecular Biology12% of their works
  4. 4Immunology3% of their works
  5. 5Cancer Research3% of their works
  6. 6Pathology and Forensic Medicine3% of their works

Research topics

Which keywords describe John A. McLachlan's research?

The keywords of their largest research topics: Environmental Exposure, Bisphenol A, Endocrine-Disrupting Chemicals, Estrogen, Receptor, Decidual NK Cells, Phytoestrogens and Regulatory T Cells.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 17 words, with their numbers
  1. Environmental Exposure1412 topics
  2. Bisphenol A1221 topic
  3. Endocrine-Disrupting Chemicals1221 topic
  4. Estrogen981 topic
  5. Receptor981 topic
  6. Decidual NK Cells211 topic
  7. Phytoestrogens211 topic
  8. Regulatory T Cells211 topic
  9. Soy Isoflavones211 topic
  10. Brominated Flame Retardants191 topic
  11. Polycyclic Aromatic Hydrocarbons191 topic
  12. Antibiotics161 topic
  13. Resistance Genes161 topic
  14. Comet Assay151 topic
  15. Genotoxicity Testing151 topic
  16. Fertility Preservation131 topic
  17. Ovarian Tissue Cryopreservation131 topic

Where does John A. McLachlan work?

John A. McLachlan's main affiliation in the publication record is Tulane University, United States.

How many publications and citations does John A. McLachlan have?

OpenAlex counts 296 works and 21,961 citations for John A. McLachlan, an h-index of 78, ranking #92,430 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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