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Researcher · Universidad de Salamanca · Spain

Juan Pedro Bolaños

Juan Pedro Bolaños publishes mostly in Molecular Biology, Physiology and Cellular and Molecular Neuroscience, on topics such as Mitochondrial Function and Pathology, Neuroscience and Neuropharmacology Research and Nitric Oxide and Endothelin Effects.

ORCID 0000-0002-3949-6862 OpenAlex
World rank
#128,151
of 7,710,434 ranked researchers
Rank in Spain
#1,749
of 204,306
Works
252
Citations
16k
Citations per work
64.6
h-index
69
i10-index 142Top 1.2% in Molecular Biology
Juan Pedro Bolaños

What does Juan Pedro Bolaños research?

Juan Pedro Bolaños's work is mostly in Biochemistry, Genetics and Molecular Biology (39%), Medicine (14%), Neuroscience (12%).

Life Sciences: 129 works · 51.0% of their workBiochemistry, Genetics and Molecular Biology: 97 works · 38.5% of their workMolecular Biology: 75 works · 29.8% of their workBiochemistry: 14 works · 5.4% of their workClinical Biochemistry: 8 works · 3.3% of their workNeuroscience: 31 works · 12.5% of their workCellular and Molecular Neuroscience: 22 works · 8.6% of their workNeurology: 10 works · 3.9% of their workEverything else: 88 works · 35.0% of their workHealth Sciences: 35 works · 14.0% of their workMedicine: 35 works · 14.0% of their workPhysiology: 35 works · 14.0% of their workTHEIR WORKJuan PedroBolaños252works
Life Sciences51%Health Sciences14%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology9738.5%
    2. Neuroscience3112.5%
  2. Everything else8835.0%
    1. Medicine3514.0%
  1. 1Molecular Biology30% of their works
  2. 2Physiology14% of their works
  3. 3Cellular and Molecular Neuroscience9% of their works
  4. 4Biochemistry5% of their works
  5. 5Neurology4% of their works
  6. 6Clinical Biochemistry3% of their works

Research topics

Which keywords describe Juan Pedro Bolaños's research?

The keywords of their largest research topics: Oxidative Stress, Reactive Oxygen Species, Metabolic Regulation, Mitochondrial Dysfunction, Cell Death, Mitochondria, Inflammation and Neurotransmission.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 23 words, with their numbers
  1. Oxidative Stress1433 topics
  2. Reactive Oxygen Species1282 topics
  3. Metabolic Regulation1062 topics
  4. Mitochondrial Dysfunction1062 topics
  5. Cell Death901 topic
  6. Mitochondria901 topic
  7. Inflammation532 topics
  8. Neurotransmission441 topic
  9. Synaptic Plasticity441 topic
  10. Endothelial Dysfunction381 topic
  11. Nitric Oxide381 topic
  12. Metabolic Disorders372 topics
  13. Mitochondrial Function372 topics
  14. Biochemical Genetics221 topic
  15. Microglia221 topic
  16. Neuroinflammation221 topic
  17. Newborn Screening221 topic
  18. Brown Adipose Tissue161 topic
  19. Thermogenesis161 topic
  20. Amino Acid Transporters151 topic
  21. D-Serine151 topic
  22. Glutathione151 topic
  23. Hydrogen Sulfide151 topic

Where does Juan Pedro Bolaños work?

Juan Pedro Bolaños's main affiliation in the publication record is Universidad de Salamanca, Spain.

How many publications and citations does Juan Pedro Bolaños have?

OpenAlex counts 252 works and 16,281 citations for Juan Pedro Bolaños, an h-index of 69, ranking #128,151 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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