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Researcher · The University of Queensland · Australia

Jürgen Götz

Jürgen Götz publishes mostly in Molecular Biology, Physiology and Cellular and Molecular Neuroscience, on topics such as Alzheimer's disease research and treatments, Ultrasound and Hyperthermia Applications and Neuroscience and Neuropharmacology Research.

ORCID 0000-0001-8501-7896 OpenAlex
World rank
#66,169
of 7,710,434 ranked researchers
Rank in Australia
#2,094
of 140,210
Works
370
Citations
29k
Citations per work
77.6
h-index
88
i10-index 221Top 0.4% in Molecular Biology
Jürgen Götz

What does Jürgen Götz research?

Jürgen Götz's work is mostly in Medicine (22%), Biochemistry, Genetics and Molecular Biology (22%), Neuroscience (14%).

Life Sciences: 134 works · 36.1% of their workBiochemistry, Genetics and Molecular Biology: 81 works · 21.8% of their workMolecular Biology: 81 works · 21.8% of their workNeuroscience: 53 works · 14.3% of their workCellular and Molecular Neuroscience: 30 works · 8.1% of their workNeurology: 23 works · 6.2% of their workEverything else: 127 works · 34.4% of their workHealth Sciences: 83 works · 22.5% of their workMedicine: 83 works · 22.5% of their workPhysiology: 68 works · 18.5% of their workNeurology: 15 works · 4.0% of their workPhysical Sciences: 26 works · 7.0% of their workEngineering: 26 works · 7.0% of their workBiomedical Engineering: 26 works · 7.0% of their workTHEIR WORKJürgen Götz370works
Life Sciences36%Physical Sciences7%Health Sciences22%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology8121.8%
    2. Neuroscience5314.3%
  2. Everything else12734.4%
    1. Medicine8322.5%
    1. Engineering267.0%
  1. 1Molecular Biology22% of their works
  2. 2Physiology18% of their works
  3. 3Cellular and Molecular Neuroscience8% of their works
  4. 4Biomedical Engineering7% of their works
  5. 5Neurology6% of their works
  6. 6Neurology4% of their works

Research topics

Which keywords describe Jürgen Götz's research?

The keywords of their largest research topics: Neurodegeneration, Neuroinflammation, Amyloid Hypothesis, Tau Pathology, Neurodegenerative Diseases, Blood-Brain Barrier, Focused Ultrasound and Neurotransmission.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 19 words, with their numbers
  1. Neurodegeneration2012 topics
  2. Neuroinflammation2012 topics
  3. Amyloid Hypothesis1721 topic
  4. Tau Pathology1721 topic
  5. Neurodegenerative Diseases983 topics
  6. Blood-Brain Barrier421 topic
  7. Focused Ultrasound421 topic
  8. Neurotransmission411 topic
  9. Synaptic Plasticity411 topic
  10. Acetylcholinesterase361 topic
  11. Cholinesterase Inhibitors361 topic
  12. Molecular Basis331 topic
  13. Prion Diseases331 topic
  14. Astrocytes291 topic
  15. Microglia291 topic
  16. Mitochondria291 topic
  17. Reactive Oxygen Species291 topic
  18. Photoacoustic Imaging191 topic
  19. Tomography191 topic

Where does Jürgen Götz work?

Jürgen Götz's main affiliation in the publication record is The University of Queensland, Australia.

How many publications and citations does Jürgen Götz have?

OpenAlex counts 370 works and 28,725 citations for Jürgen Götz, an h-index of 88, ranking #66,169 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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