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Researcher · CHDI Foundation · United States

Longbin Liu

Longbin Liu publishes mostly in Cellular and Molecular Neuroscience, Molecular Biology and Neurology, on topics such as Genetic Neurodegenerative Diseases, Mitochondrial Function and Pathology and Neurological disorders and treatments.

ORCID 0000-0003-3995-0196 OpenAlex
World rank
#3,903,956
of 7,710,434 ranked researchers
Rank in United States
#1,070,081
of 1,627,098
Works
32
Citations
193
Citations per work
6.0
h-index
6
i10-index 4Top 95% in Cellular and Molecular Neuroscience
Longbin Liu

What does Longbin Liu research?

Longbin Liu's work is mostly in Neuroscience (31%), Medicine (26%), Biochemistry, Genetics and Molecular Biology (24%).

Life Sciences: 19 works · 60.0% of their workNeuroscience: 10 works · 31.4% of their workCellular and Molecular Neuroscience: 10 works · 31.4% of their workBiochemistry, Genetics and Molecular Biology: 8 works · 24.3% of their workMolecular Biology: 6 works · 20.0% of their workCell Biology: 1 works · 4.3% of their workPharmacology, Toxicology and Pharmaceutics: 1 works · 4.3% of their workPharmaceutical Science: 1 works · 4.3% of their workHealth Sciences: 8 works · 25.7% of their workMedicine: 8 works · 25.7% of their workNeurology: 5 works · 15.7% of their workRadiology, Nuclear Medicine and Imaging: 3 works · 10.0% of their workEverything else: 5 works · 14.3% of their workTHEIR WORKLongbin Liu32works
Life Sciences60%Health Sciences26%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Neuroscience1031.4%
    2. Biochemistry, Genetics and Molecular Biology824.3%
    3. Pharmacology, Toxicology and Pharmaceutics14.3%
    1. Medicine825.7%
  3. Everything else514.3%
  1. 1Cellular and Molecular Neuroscience31% of their works
  2. 2Molecular Biology20% of their works
  3. 3Neurology16% of their works
  4. 4Radiology, Nuclear Medicine and Imaging10% of their works
  5. 5Cell Biology4% of their works
  6. 6Pharmaceutical Science4% of their works

Research topics

Which keywords describe Longbin Liu's research?

The keywords of their largest research topics: Mitochondrial Dysfunction, Huntington's Disease, Protein Aggregation, Mitochondria, Neurological Disorders, Reactive Oxygen Species, Deep Brain Stimulation and Parkinson's Disease.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 18 words, with their numbers
  1. Mitochondrial Dysfunction292 topics
  2. Huntington's Disease191 topic
  3. Protein Aggregation191 topic
  4. Mitochondria101 topic
  5. Neurological Disorders102 topics
  6. Reactive Oxygen Species101 topic
  7. Deep Brain Stimulation81 topic
  8. Parkinson's Disease81 topic
  9. Imaging51 topic
  10. PET/CT51 topic
  11. FUS/TLS31 topic
  12. Fluorine31 topic
  13. Medicinal Chemistry31 topic
  14. TDP-4331 topic
  15. Endocytosis21 topic
  16. Membrane Fusion21 topic
  17. Neurotransmission21 topic
  18. Synaptic Plasticity21 topic

Where does Longbin Liu work?

Longbin Liu's main affiliation in the publication record is CHDI Foundation, United States.

How many publications and citations does Longbin Liu have?

OpenAlex counts 32 works and 193 citations for Longbin Liu, an h-index of 6, ranking #3,903,956 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Other researchers named Longbin Liu

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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