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Researcher · MediciNova (United States) · United States

Malath M. Makhay

Malath M. Makhay publishes mostly in Cellular and Molecular Neuroscience, Molecular Biology and Animal Science and Zoology, on topics such as Neurotransmitter Receptor Influence on Behavior, Receptor Mechanisms and Signaling and Neuroscience and Neuropharmacology Research.

OpenAlex
World rank
#5,392,118
of 7,710,434 ranked researchers
Rank in United States
#1,368,169
of 1,627,098
Works
25
Citations
552
Citations per work
22.1
h-index
10
i10-index 10Top 79% in Cellular and Molecular Neuroscience
Malath M. Makhay

What does Malath M. Makhay research?

Malath M. Makhay's work is mostly in Neuroscience (30%), Biochemistry, Genetics and Molecular Biology (16%), Medicine (14%).

Life Sciences: 15 works · 58.6% of their workNeuroscience: 8 works · 30.0% of their workCellular and Molecular Neuroscience: 8 works · 30.0% of their workBiochemistry, Genetics and Molecular Biology: 4 works · 15.7% of their workMolecular Biology: 4 works · 15.7% of their workAgricultural and Biological Sciences: 2 works · 8.6% of their workAnimal Science and Zoology: 2 works · 8.6% of their workImmunology and Microbiology: 1 works · 4.3% of their workImmunology: 1 works · 4.3% of their workEverything else: 7 works · 27.1% of their workHealth Sciences: 4 works · 14.3% of their workMedicine: 4 works · 14.3% of their workPulmonary and Respiratory Medicine: 2 works · 8.6% of their workPhysiology: 1 works · 5.7% of their workTHEIR WORKMalath M. Makhay25works
Life Sciences59%Health Sciences14%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Neuroscience830.0%
    2. Biochemistry, Genetics and Molecular Biology415.7%
    3. Agricultural and Biological Sciences28.6%
    4. Immunology and Microbiology14.3%
  2. Everything else727.1%
    1. Medicine414.3%
  1. 1Cellular and Molecular Neuroscience30% of their works
  2. 2Molecular Biology16% of their works
  3. 3Animal Science and Zoology9% of their works
  4. 4Pulmonary and Respiratory Medicine9% of their works
  5. 5Physiology6% of their works
  6. 6Immunology4% of their works

Research topics

Which keywords describe Malath M. Makhay's research?

The keywords of their largest research topics: Stress, Dopamine, Serotonin, Crystal Structure, Depression, G-Protein-Coupled Receptors, Neurotransmission and Synaptic Plasticity.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 18 words, with their numbers
  1. Stress173 topics
  2. Dopamine101 topic
  3. Serotonin101 topic
  4. Crystal Structure71 topic
  5. Depression72 topics
  6. G-Protein-Coupled Receptors71 topic
  7. Neurotransmission61 topic
  8. Synaptic Plasticity61 topic
  9. Clenbuterol41 topic
  10. Neuropeptides41 topic
  11. Ractopamine41 topic
  12. Receptors41 topic
  13. Brain31 topic
  14. Cortisol31 topic
  15. Fibrosis31 topic
  16. Idiopathic Pulmonary Fibrosis31 topic
  17. Inflammatory Response31 topic
  18. Innate Immunity31 topic

Where does Malath M. Makhay work?

Malath M. Makhay's main affiliation in the publication record is MediciNova (United States), United States.

How many publications and citations does Malath M. Makhay have?

OpenAlex counts 25 works and 552 citations for Malath M. Makhay, an h-index of 10, ranking #5,392,118 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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