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Researcher · CHDI Foundation · United States

Maria G. Beconi

Maria G. Beconi publishes mostly in Cellular and Molecular Neuroscience, Molecular Biology and Oncology, on topics such as Genetic Neurodegenerative Diseases, Diabetes Treatment and Management and Peptidase Inhibition and Analysis.

OpenAlex
World rank
#1,774,872
of 7,710,434 ranked researchers
Rank in United States
#546,290
of 1,627,098
Works
43
Citations
2.8k
Citations per work
64.4
h-index
23
i10-index 31Top 28% in Cellular and Molecular Neuroscience
Maria G. Beconi

What does Maria G. Beconi research?

Maria G. Beconi's work is mostly in Medicine (18%), Biochemistry, Genetics and Molecular Biology (17%), Neuroscience (17%).

Everything else: 19 works · 44.4% of their workLife Sciences: 16 works · 37.8% of their workBiochemistry, Genetics and Molecular Biology: 7 works · 16.9% of their workMolecular Biology: 7 works · 16.9% of their workNeuroscience: 7 works · 16.9% of their workCellular and Molecular Neuroscience: 7 works · 16.9% of their workPharmacology, Toxicology and Pharmaceutics: 2 works · 4.0% of their workPharmacology: 2 works · 4.0% of their workHealth Sciences: 8 works · 17.8% of their workMedicine: 8 works · 17.8% of their workOncology: 3 works · 7.3% of their workEndocrinology, Diabetes and Metabolism: 3 works · 6.5% of their workPhysiology: 2 works · 4.0% of their workTHEIR WORKMaria G. Beconi43works
Life Sciences38%Health Sciences18%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
  1. Everything else1944.4%
    1. Biochemistry, Genetics and Molecular Biology716.9%
    2. Neuroscience716.9%
    3. Pharmacology, Toxicology and Pharmaceutics24.0%
    1. Medicine817.8%
  1. 1Cellular and Molecular Neuroscience17% of their works
  2. 2Molecular Biology17% of their works
  3. 3Oncology7% of their works
  4. 4Endocrinology, Diabetes and Metabolism6% of their works
  5. 5Physiology4% of their works
  6. 6Pharmacology4% of their works

Research topics

Which keywords describe Maria G. Beconi's research?

The keywords of their largest research topics: Mitochondrial Dysfunction, Depression, Cardiovascular Outcomes, Fibroblast Activation Protein, Huntington's Disease, Protein Aggregation, Tumor Microenvironment and Type 2 Diabetes.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 19 words, with their numbers
  1. Mitochondrial Dysfunction122 topics
  2. Depression102 topics
  3. Cardiovascular Outcomes81 topic
  4. Fibroblast Activation Protein81 topic
  5. Huntington's Disease81 topic
  6. Protein Aggregation81 topic
  7. Tumor Microenvironment81 topic
  8. Type 2 Diabetes81 topic
  9. Cytochrome P45072 topics
  10. Neuropeptides61 topic
  11. Receptors61 topic
  12. Brain Iron Accumulation41 topic
  13. Cytokines41 topic
  14. Drug Metabolism41 topic
  15. Neurodegeneration41 topic
  16. Neuroinflammation41 topic
  17. Pharmacogenetics41 topic
  18. Arachidonic Acid31 topic
  19. Epoxyeicosatrienoic Acids31 topic

Where does Maria G. Beconi work?

Maria G. Beconi's main affiliation in the publication record is CHDI Foundation, United States.

How many publications and citations does Maria G. Beconi have?

OpenAlex counts 43 works and 2,769 citations for Maria G. Beconi, an h-index of 23, ranking #1,774,872 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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