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Researcher · University of California, Los Angeles · United States

Michael V. Sofroniew

Michael V. Sofroniew publishes mostly in Cellular and Molecular Neuroscience, Molecular Biology and Neurology, on topics such as Neurogenesis and neuroplasticity mechanisms, Neuroinflammation and Neurodegeneration Mechanisms and Nerve injury and regeneration.

ORCID 0000-0001-6075-0178 OpenAlex
World rank
#11,695
of 7,710,434 ranked researchers
Rank in United States
#4,040
of 1,627,098
Works
248
Citations
53k
Citations per work
213.5
h-index
99
i10-index 186Top 0.4% in Cellular and Molecular Neuroscience
Michael V. Sofroniew

What does Michael V. Sofroniew research?

Michael V. Sofroniew's work is mostly in Neuroscience (45%), Biochemistry, Genetics and Molecular Biology (14%), Psychology (4%).

Life Sciences: 147 works · 59.2% of their workNeuroscience: 112 works · 45.2% of their workCellular and Molecular Neuroscience: 48 works · 19.3% of their workNeurology: 29 works · 11.6% of their workDevelopmental Neuroscience: 26 works · 10.5% of their workEndocrine and Autonomic Systems: 9 works · 3.8% of their workBiochemistry, Genetics and Molecular Biology: 35 works · 14.0% of their workMolecular Biology: 35 works · 14.0% of their workEverything else: 92 works · 37.0% of their workSocial Sciences: 9 works · 3.8% of their workPsychology: 9 works · 3.8% of their workSocial Psychology: 9 works · 3.8% of their workTHEIR WORKMichael V.Sofroniew248works
Life Sciences59%Social Sciences4%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Neuroscience11245.2%
    2. Biochemistry, Genetics and Molecular Biology3514.0%
  2. Everything else9237.0%
    1. Psychology93.8%
  1. 1Cellular and Molecular Neuroscience19% of their works
  2. 2Molecular Biology14% of their works
  3. 3Neurology12% of their works
  4. 4Developmental Neuroscience10% of their works
  5. 5Social Psychology4% of their works
  6. 6Endocrine and Autonomic Systems4% of their works

Research topics

Which keywords describe Michael V. Sofroniew's research?

The keywords of their largest research topics: Brain Development, Synaptic Plasticity, Depression, Adult Stem Cells, Neurogenesis, Microglia, Neuroinflammation and BDNF.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 23 words, with their numbers
  1. Brain Development1352 topics
  2. Synaptic Plasticity922 topics
  3. Depression853 topics
  4. Adult Stem Cells701 topic
  5. Neurogenesis701 topic
  6. Microglia651 topic
  7. Neuroinflammation651 topic
  8. BDNF531 topic
  9. Neurotrophins531 topic
  10. Anxiety452 topics
  11. Neuropeptides452 topics
  12. Neurobiology412 topics
  13. GABAergic391 topic
  14. Neurotransmission391 topic
  15. Stress322 topics
  16. Oxytocin271 topic
  17. Social Behavior271 topic
  18. Opioid181 topic
  19. Receptors181 topic
  20. Central Chemoreception171 topic
  21. Respiratory Rhythm171 topic
  22. Brain141 topic
  23. Cortisol141 topic

Where does Michael V. Sofroniew work?

Michael V. Sofroniew's main affiliation in the publication record is University of California, Los Angeles, United States.

How many publications and citations does Michael V. Sofroniew have?

OpenAlex counts 248 works and 52,940 citations for Michael V. Sofroniew, an h-index of 99, ranking #11,695 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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