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Researcher · Temple University · United States

Muniswamy Madesh

Muniswamy Madesh publishes mostly in Molecular Biology, Physiology and Cardiology and Cardiovascular Medicine, on topics such as Mitochondrial Function and Pathology, ATP Synthase and ATPases Research and Ion Channels and Receptors.

ORCID 0000-0001-6745-9092 OpenAlex
World rank
#54,447
of 7,710,434 ranked researchers
Rank in United States
#21,052
of 1,627,098
Works
248
Citations
26k
Citations per work
106.2
h-index
74
i10-index 167Top 0.9% in Molecular Biology
Muniswamy Madesh

What does Muniswamy Madesh research?

Muniswamy Madesh's work is mostly in Biochemistry, Genetics and Molecular Biology (38%), Medicine (16%), Neuroscience (4%).

Life Sciences: 103 works · 41.5% of their workBiochemistry, Genetics and Molecular Biology: 93 works · 37.7% of their workMolecular Biology: 93 works · 37.7% of their workNeuroscience: 9 works · 3.8% of their workSensory Systems: 9 works · 3.8% of their workEverything else: 95 works · 38.4% of their workHealth Sciences: 50 works · 20.1% of their workMedicine: 40 works · 16.3% of their workPhysiology: 19 works · 7.7% of their workCardiology and Cardiovascular Medicine: 11 works · 4.5% of their workEpidemiology: 10 works · 4.1% of their workNursing: 9 works · 3.8% of their workNutrition and Dietetics: 9 works · 3.8% of their workTHEIR WORKMuniswamy Madesh248works
Life Sciences41%Health Sciences20%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology9337.7%
    2. Neuroscience93.8%
  2. Everything else9538.4%
    1. Medicine4016.3%
    2. Nursing93.8%
  1. 1Molecular Biology38% of their works
  2. 2Physiology8% of their works
  3. 3Cardiology and Cardiovascular Medicine4% of their works
  4. 4Epidemiology4% of their works
  5. 5Sensory Systems4% of their works
  6. 6Nutrition and Dietetics4% of their works

Research topics

Which keywords describe Muniswamy Madesh's research?

The keywords of their largest research topics: Reactive Oxygen Species, Oxidative Stress, Cell Death, Mitochondria, Mitochondrial Dysfunction, Mitochondrial Fusion, Ion Channels and Inflammation.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 23 words, with their numbers
  1. Reactive Oxygen Species1404 topics
  2. Oxidative Stress1213 topics
  3. Cell Death1012 topics
  4. Mitochondria1012 topics
  5. Mitochondrial Dysfunction821 topic
  6. Mitochondrial Fusion821 topic
  7. Ion Channels482 topics
  8. Inflammation442 topics
  9. Apoptosis392 topics
  10. ATP Synthase381 topic
  11. Vacuolar ATPases381 topic
  12. Calcium Signaling251 topic
  13. TRP Channels251 topic
  14. Endothelial Dysfunction241 topic
  15. Nitric Oxide241 topic
  16. Calcium Channels231 topic
  17. Voltage-Gated Channels231 topic
  18. Autophagy201 topic
  19. Disease201 topic
  20. Bcl-2 Family191 topic
  21. TNF Signaling191 topic
  22. Cardioprotection151 topic
  23. Ischemia-Reperfusion Injury151 topic

Where does Muniswamy Madesh work?

Muniswamy Madesh's main affiliation in the publication record is Temple University, United States.

How many publications and citations does Muniswamy Madesh have?

OpenAlex counts 248 works and 26,349 citations for Muniswamy Madesh, an h-index of 74, ranking #54,447 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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