Science Explorer Back to mapMap
Discussion
Researcher · Murdoch Children's Research Institute · Australia

Nadia Mazarakis

Nadia Mazarakis publishes mostly in Infectious Diseases, Molecular Biology and Immunology, on topics such as SARS-CoV-2 and COVID-19 Research, vaccines and immunoinformatics approaches and Immune responses and vaccinations.

ORCID 0000-0001-7681-2775 OpenAlex
World rank
#2,215,338
of 7,710,434 ranked researchers
Rank in Australia
#63,848
of 140,210
Works
45
Citations
422
Citations per work
9.4
h-index
10
i10-index 12Top 64% in Infectious Diseases
Nadia Mazarakis

What does Nadia Mazarakis research?

Nadia Mazarakis's work is mostly in Medicine (43%), Immunology and Microbiology (18%), Biochemistry, Genetics and Molecular Biology (17%).

Health Sciences: 19 works · 42.6% of their workMedicine: 19 works · 42.6% of their workInfectious Diseases: 16 works · 35.2% of their workEpidemiology: 3 works · 7.4% of their workLife Sciences: 16 works · 35.3% of their workImmunology and Microbiology: 8 works · 18.1% of their workImmunology: 7 works · 14.8% of their workMicrobiology: 1 works · 3.3% of their workBiochemistry, Genetics and Molecular Biology: 8 works · 17.2% of their workMolecular Biology: 8 works · 17.2% of their workEverything else: 9 works · 19.6% of their workSocial Sciences: 1 works · 2.5% of their workSocial Sciences: 1 works · 2.5% of their workHealth: 1 works · 2.5% of their workTHEIR WORKNadia Mazarakis45works
Life Sciences35%Health Sciences43%Social Sciences2%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Medicine1942.6%
    1. Immunology and Microbiology818.1%
    2. Biochemistry, Genetics and Molecular Biology817.2%
  3. Everything else919.6%
    1. Social Sciences12.5%
  1. 1Infectious Diseases35% of their works
  2. 2Molecular Biology17% of their works
  3. 3Immunology15% of their works
  4. 4Epidemiology7% of their works
  5. 5Microbiology3% of their works
  6. 6Health2% of their works

Research topics

Which keywords describe Nadia Mazarakis's research?

The keywords of their largest research topics: SARS-CoV-2, ACE2 Receptor, Pandemic, Coronavirus, Vaccine, antibody, spike protein and Epidemiology.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most.

All 23 words, with their numbers
  1. SARS-CoV-2413 topics
  2. ACE2 Receptor342 topics
  3. Pandemic342 topics
  4. Coronavirus292 topics
  5. Vaccine292 topics
  6. antibody261 topic
  7. spike protein261 topic
  8. Epidemiology112 topics
  9. Immune Response112 topics
  10. Epitope Prediction101 topic
  11. Peptide-MHC Binding101 topic
  12. Clinical Characteristics81 topic
  13. Innate Memory81 topic
  14. Thrombosis81 topic
  15. Trained Immunity81 topic
  16. COVID-1971 topic
  17. Diagnostics71 topic
  18. Nrf271 topic
  19. Oxidative Stress71 topic
  20. IL-3341 topic
  21. Innate Lymphoid Cells41 topic
  22. Circovirus31 topic
  23. Porcine Reproductive and Respiratory Syndrome Virus31 topic

Where does Nadia Mazarakis work?

Nadia Mazarakis's main affiliation in the publication record is Murdoch Children's Research Institute, Australia.

How many publications and citations does Nadia Mazarakis have?

OpenAlex counts 45 works and 422 citations for Nadia Mazarakis, an h-index of 10, ranking #2,215,338 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

Ask a question or share a note about Nadia Mazarakis. Posts are plain text and can be reported.