Science Explorer Back to mapMap
Discussion
Researcher · University of Birmingham · United Kingdom

Philip Noel Newsome

Philip Noel Newsome publishes mostly in Epidemiology, Hepatology and Endocrinology, Diabetes and Metabolism, on topics such as Liver Disease Diagnosis and Treatment, Liver Disease and Transplantation and Liver physiology and pathology.

ORCID 0000-0001-6085-3652 OpenAlex
World rank
#63,131
of 7,710,434 ranked researchers
Rank in United Kingdom
#5,337
of 367,258
Works
560
Citations
52k
Citations per work
92.8
h-index
84
i10-index 220Top 0.4% in Epidemiology
Philip Noel Newsome

What does Philip Noel Newsome research?

Philip Noel Newsome's work is mostly in Medicine (63%), Biochemistry, Genetics and Molecular Biology (5%).

Health Sciences: 353 works · 63.0% of their workMedicine: 353 works · 63.0% of their workEpidemiology: 119 works · 21.3% of their workHepatology: 114 works · 20.3% of their workEndocrinology, Diabetes and Metabolism: 49 works · 8.8% of their workSurgery: 49 works · 8.7% of their workGenetics: 22 works · 3.9% of their workEverything else: 178 works · 31.8% of their workLife Sciences: 29 works · 5.2% of their workBiochemistry, Genetics and Molecular Biology: 29 works · 5.2% of their workMolecular Biology: 29 works · 5.2% of their workTHEIR WORKPhilip NoelNewsome560works
Life Sciences5%Health Sciences63%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Medicine35363.0%
  2. Everything else17831.8%
    1. Biochemistry, Genetics and Molecular Biology295.2%
  1. 1Epidemiology21% of their works
  2. 2Hepatology20% of their works
  3. 3Endocrinology, Diabetes and Metabolism9% of their works
  4. 4Surgery9% of their works
  5. 5Molecular Biology5% of their works
  6. 6Genetics4% of their works

Research topics

Which keywords describe Philip Noel Newsome's research?

The keywords of their largest research topics: Insulin Resistance, Metabolic Syndrome, NAFLD, NASH, Inflammation, Oxidative Stress, Hepatic Encephalopathy and Liver Cirrhosis.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 20 words, with their numbers
  1. Insulin Resistance3452 topics
  2. Metabolic Syndrome3452 topics
  3. NAFLD2941 topic
  4. NASH2941 topic
  5. Inflammation1262 topics
  6. Oxidative Stress1262 topics
  7. Hepatic Encephalopathy1151 topic
  8. Liver Cirrhosis1151 topic
  9. Hepatic Stellate Cells891 topic
  10. Liver Regeneration891 topic
  11. Mesenchymal Stem Cells571 topic
  12. Multipotent571 topic
  13. Fructose511 topic
  14. Obesity511 topic
  15. Autoimmune Hepatitis461 topic
  16. Primary Biliary Cirrhosis461 topic
  17. Alcohol371 topic
  18. Liver Disease371 topic
  19. Cardiovascular Outcomes361 topic
  20. Type 2 Diabetes361 topic

Where does Philip Noel Newsome work?

Philip Noel Newsome's main affiliation in the publication record is University of Birmingham, United Kingdom.

How many publications and citations does Philip Noel Newsome have?

OpenAlex counts 560 works and 51,972 citations for Philip Noel Newsome, an h-index of 84, ranking #63,131 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

Ask a question or share a note about Philip Noel Newsome. Keep it to the research: no personal attacks, advertising or links. Any reader can report a post, which collapses it for everyone until it is opened.