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Researcher · University of Wisconsin–Madison · United States

Richard H. Weindruch

Richard H. Weindruch publishes mostly in Physiology, Aging and Molecular Biology, on topics such as Genetics, Aging, and Longevity in Model Organisms, Adipose Tissue and Metabolism and Diet and metabolism studies.

OpenAlex
World rank
#154,244
of 7,710,434 ranked researchers
Rank in United States
#58,094
of 1,627,098
Works
258
Citations
33k
Citations per work
128.0
h-index
84
i10-index 189Top 0.7% in Physiology (Medicine)
Richard H. Weindruch

What does Richard H. Weindruch research?

Richard H. Weindruch's work is mostly in Biochemistry, Genetics and Molecular Biology (33%), Medicine (32%), Neuroscience (5%).

Life Sciences: 99 works · 38.5% of their workBiochemistry, Genetics and Molecular Biology: 86 works · 33.3% of their workAging: 46 works · 17.9% of their workMolecular Biology: 40 works · 15.4% of their workNeuroscience: 13 works · 5.2% of their workEndocrine and Autonomic Systems: 8 works · 3.2% of their workNeurology: 5 works · 2.0% of their workHealth Sciences: 82 works · 31.9% of their workMedicine: 82 works · 31.9% of their workPhysiology: 74 works · 28.7% of their workPediatrics, Perinatology and Child Health: 8 works · 3.2% of their workEverything else: 76 works · 29.6% of their workTHEIR WORKRichard H.Weindruch258works
Life Sciences38%Health Sciences32%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology8633.3%
    2. Neuroscience135.2%
    1. Medicine8231.9%
  3. Everything else7629.6%
  1. 1Physiology29% of their works
  2. 2Aging18% of their works
  3. 3Molecular Biology15% of their works
  4. 4Pediatrics, Perinatology and Child Health3% of their works
  5. 5Endocrine and Autonomic Systems3% of their works
  6. 6Neurology2% of their works

Research topics

Which keywords describe Richard H. Weindruch's research?

The keywords of their largest research topics: Aging, Oxidative Stress, Genetic Regulation, Longevity, Metabolic Regulation, Mitochondrial Dysfunction, Inflammation and Weight Loss.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 23 words, with their numbers
  1. Aging2003 topics
  2. Oxidative Stress1773 topics
  3. Genetic Regulation1191 topic
  4. Longevity1191 topic
  5. Metabolic Regulation1162 topics
  6. Mitochondrial Dysfunction1162 topics
  7. Inflammation852 topics
  8. Weight Loss852 topics
  9. Brown Adipose Tissue721 topic
  10. Thermogenesis721 topic
  11. Metabolic Syndrome692 topics
  12. Ketogenic Diet481 topic
  13. Low-Carbohydrate Diet481 topic
  14. Mitochondria441 topic
  15. Reactive Oxygen Species441 topic
  16. Intermittent Fasting371 topic
  17. Time-Restricted Feeding371 topic
  18. Early-Life Conditions211 topic
  19. Fetal Programming211 topic
  20. Muscle Regeneration151 topic
  21. Skeletal Muscle Atrophy151 topic
  22. Circadian Clock141 topic
  23. Melatonin141 topic

Where does Richard H. Weindruch work?

Richard H. Weindruch's main affiliation in the publication record is University of Wisconsin–Madison, United States.

How many publications and citations does Richard H. Weindruch have?

OpenAlex counts 258 works and 33,025 citations for Richard H. Weindruch, an h-index of 84, ranking #154,244 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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