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Researcher · Mie University · Japan

Shin‐ichiro Takebayashi

Shin‐ichiro Takebayashi publishes mostly in Molecular Biology, Genetics and Biomedical Engineering, on topics such as Epigenetics and DNA Methylation, Genomics and Chromatin Dynamics and DNA Repair Mechanisms.

ORCID 0000-0002-1968-9800 OpenAlex
World rank
#1,203,521
of 7,710,434 ranked researchers
Rank in Japan
#17,522
of 383,136
Works
69
Citations
4.3k
Citations per work
62.7
h-index
25
i10-index 38Top 22% in Molecular Biology
Shin‐ichiro Takebayashi

What does Shin‐ichiro Takebayashi research?

Shin‐ichiro Takebayashi's work is mostly in Biochemistry, Genetics and Molecular Biology (69%), Engineering (4%), Agricultural and Biological Sciences (4%).

Life Sciences: 50 works · 72.5% of their workBiochemistry, Genetics and Molecular Biology: 48 works · 69.0% of their workMolecular Biology: 39 works · 56.7% of their workGenetics: 6 works · 8.2% of their workCancer Research: 3 works · 4.1% of their workAgricultural and Biological Sciences: 2 works · 3.5% of their workPlant Science: 2 works · 3.5% of their workEverything else: 15 works · 21.1% of their workPhysical Sciences: 3 works · 4.1% of their workEngineering: 3 works · 4.1% of their workBiomedical Engineering: 3 works · 4.1% of their workHealth Sciences: 2 works · 2.3% of their workMedicine: 2 works · 2.3% of their workPhysiology: 2 works · 2.3% of their workTHEIR WORKShin‐ichiroTakebayashi69works
Life Sciences72%Physical Sciences4%Health Sciences2%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology4869.0%
    2. Agricultural and Biological Sciences23.5%
  2. Everything else1521.1%
    1. Engineering34.1%
    1. Medicine22.3%
  1. 1Molecular Biology57% of their works
  2. 2Genetics8% of their works
  3. 3Biomedical Engineering4% of their works
  4. 4Cancer Research4% of their works
  5. 5Plant Science4% of their works
  6. 6Physiology2% of their works

Research topics

Which keywords describe Shin‐ichiro Takebayashi's research?

The keywords of their largest research topics: Chromatin, DNA Methylation, Epigenetic Reprogramming, Genomic Instability, Histone Modification, Histone Modifications, Transcription and DNA Damage.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 21 words, with their numbers
  1. Chromatin362 topics
  2. DNA Methylation272 topics
  3. Epigenetic Reprogramming221 topic
  4. Genomic Instability222 topics
  5. Histone Modification221 topic
  6. Histone Modifications201 topic
  7. Transcription201 topic
  8. DNA Damage161 topic
  9. Repair161 topic
  10. Gene Expression122 topics
  11. Epigenetic Regulation92 topics
  12. Single-Cell71 topic
  13. Transcriptomics71 topic
  14. Cancer Genomics61 topic
  15. Mutational Signatures61 topic
  16. Genomic Imprinting51 topic
  17. Parental Influence51 topic
  18. Copy Number Variation41 topic
  19. Genome Evolution41 topic
  20. Genomic Rearrangements41 topic
  21. Polyploidy41 topic

Where does Shin‐ichiro Takebayashi work?

Shin‐ichiro Takebayashi's main affiliation in the publication record is Mie University, Japan.

How many publications and citations does Shin‐ichiro Takebayashi have?

OpenAlex counts 69 works and 4,323 citations for Shin‐ichiro Takebayashi, an h-index of 25, ranking #1,203,521 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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