Science Explorer Back to mapMap
Discussion
Researcher · Eisai (United Kingdom) · United Kingdom

Stephen J. Neame

Stephen J. Neame publishes mostly in Molecular Biology, Immunology and Cellular and Molecular Neuroscience, on topics such as Cell death mechanisms and regulation, Neuroscience and Neuropharmacology Research and Inflammation biomarkers and pathways.

ORCID 0000-0002-6951-032X OpenAlex
World rank
#2,166,444
of 7,710,434 ranked researchers
Rank in United Kingdom
#157,059
of 367,258
Works
23
Citations
1.6k
Citations per work
70.6
h-index
12
i10-index 13Top 65% in Molecular Biology
Stephen J. Neame

What does Stephen J. Neame research?

Stephen J. Neame's work is mostly in Biochemistry, Genetics and Molecular Biology (54%), Neuroscience (18%), Immunology and Microbiology (11%).

Life Sciences: 19 works · 83.1% of their workBiochemistry, Genetics and Molecular Biology: 12 works · 53.8% of their workMolecular Biology: 9 works · 40.0% of their workCell Biology: 2 works · 9.2% of their workCancer Research: 1 works · 4.6% of their workNeuroscience: 4 works · 18.5% of their workCellular and Molecular Neuroscience: 2 works · 10.8% of their workNeurology: 2 works · 7.7% of their workImmunology and Microbiology: 2 works · 10.8% of their workImmunology: 2 works · 10.8% of their workEverything else: 4 works · 16.9% of their workTHEIR WORKStephen J. Neame23works
Life Sciences83%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology1253.8%
    2. Neuroscience418.5%
    3. Immunology and Microbiology210.8%
  2. Everything else416.9%
  1. 1Molecular Biology40% of their works
  2. 2Immunology11% of their works
  3. 3Cellular and Molecular Neuroscience11% of their works
  4. 4Cell Biology9% of their works
  5. 5Neurology8% of their works
  6. 6Cancer Research5% of their works

Research topics

Which keywords describe Stephen J. Neame's research?

The keywords of their largest research topics: Cell Death, Mitochondria, Synaptic Plasticity, Inflammation, Angiogenesis, Apoptosis, Bcl-2 Family and Cell Signaling.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 22 words, with their numbers
  1. Cell Death102 topics
  2. Mitochondria102 topics
  3. Synaptic Plasticity102 topics
  4. Inflammation92 topics
  5. Angiogenesis72 topics
  6. Apoptosis71 topic
  7. Bcl-2 Family71 topic
  8. Cell Signaling72 topics
  9. GABAergic61 topic
  10. Neurotransmission61 topic
  11. Endocan51 topic
  12. Microglia51 topic
  13. Neuroinflammation51 topic
  14. TREM-151 topic
  15. Glycans41 topic
  16. Glycosylation41 topic
  17. Hyaluronan41 topic
  18. Proteoglycans41 topic
  19. Matrix Metalloproteinases31 topic
  20. Mitochondrial Dysfunction31 topic
  21. Oxidative Stress31 topic
  22. Tissue Inhibitors of Metalloproteinases31 topic

Where does Stephen J. Neame work?

Stephen J. Neame's main affiliation in the publication record is Eisai (United Kingdom), United Kingdom.

How many publications and citations does Stephen J. Neame have?

OpenAlex counts 23 works and 1,624 citations for Stephen J. Neame, an h-index of 12, ranking #2,166,444 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

Ask a question or share a note about Stephen J. Neame. Keep it to the research: no personal attacks, advertising or links. Any reader can report a post, which collapses it for everyone until it is opened.