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Researcher · Shujitsu University · Japan

Toshiro Niwa

Toshiro Niwa publishes mostly in Pharmacology, Oncology and Molecular Biology, on topics such as Pharmacogenetics and Drug Metabolism, Drug Transport and Resistance Mechanisms and Eicosanoids and Hypertension Pharmacology.

ORCID 0000-0002-1441-6977 OpenAlex
World rank
#4,103,886
of 7,710,434 ranked researchers
Rank in Japan
#111,619
of 383,136
Works
133
Citations
2.8k
Citations per work
21.1
h-index
28
i10-index 64Top 9.6% in Pharmacology (Pharmacology, Toxicology and Pharmaceutics)
Toshiro Niwa

What does Toshiro Niwa research?

Toshiro Niwa's work is mostly in Pharmacology, Toxicology and Pharmaceutics (25%), Medicine (14%), Biochemistry, Genetics and Molecular Biology (13%).

Everything else: 55 works · 41.3% of their workLife Sciences: 52 works · 38.8% of their workPharmacology, Toxicology and Pharmaceutics: 34 works · 25.3% of their workPharmacology: 34 works · 25.3% of their workBiochemistry, Genetics and Molecular Biology: 18 works · 13.5% of their workMolecular Biology: 10 works · 7.3% of their workBiochemistry: 8 works · 6.2% of their workHealth Sciences: 19 works · 14.5% of their workMedicine: 19 works · 14.5% of their workOncology: 11 works · 8.6% of their workEndocrinology, Diabetes and Metabolism: 8 works · 5.9% of their workPhysical Sciences: 7 works · 5.4% of their workChemistry: 7 works · 5.4% of their workOrganic Chemistry: 7 works · 5.4% of their workTHEIR WORKToshiro Niwa133works
Life Sciences39%Physical Sciences5%Health Sciences14%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
  1. Everything else5541.3%
    1. Pharmacology, Toxicology and Pharmaceutics3425.3%
    2. Biochemistry, Genetics and Molecular Biology1813.5%
    1. Medicine1914.5%
    1. Chemistry75.4%
  1. 1Pharmacology25% of their works
  2. 2Oncology9% of their works
  3. 3Molecular Biology7% of their works
  4. 4Biochemistry6% of their works
  5. 5Endocrinology, Diabetes and Metabolism6% of their works
  6. 6Organic Chemistry5% of their works

Research topics

Which keywords describe Toshiro Niwa's research?

The keywords of their largest research topics: Cytochrome P450, Pharmacokinetics, Drug Metabolism, Pharmacogenetics, Hypertension, ATP-dependent Transporters, Multidrug Resistance and Arachidonic Acid.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 19 words, with their numbers
  1. Cytochrome P4501012 topics
  2. Pharmacokinetics902 topics
  3. Drug Metabolism821 topic
  4. Pharmacogenetics821 topic
  5. Hypertension322 topics
  6. ATP-dependent Transporters271 topic
  7. Multidrug Resistance271 topic
  8. Arachidonic Acid191 topic
  9. Epoxyeicosatrienoic Acids191 topic
  10. Comet Assay161 topic
  11. Genotoxicity Testing161 topic
  12. Aldosterone131 topic
  13. Primary Aldosteronism131 topic
  14. Chiral Separation111 topic
  15. Liquid Chromatography111 topic
  16. Antiepileptic Drugs91 topic
  17. Prenatal Exposure91 topic
  18. Molecular Docking81 topic
  19. Virtual Screening81 topic

Where does Toshiro Niwa work?

Toshiro Niwa's main affiliation in the publication record is Shujitsu University, Japan.

How many publications and citations does Toshiro Niwa have?

OpenAlex counts 133 works and 2,806 citations for Toshiro Niwa, an h-index of 28, ranking #4,103,886 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

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