Science Explorer Back to mapMap
Discussion
Researcher · Thrombosis Research Institute · United Kingdom

Vincent Ellis

Vincent Ellis publishes mostly in Cancer Research, Hematology and Molecular Biology, on topics such as Protease and Inhibitor Mechanisms, Blood Coagulation and Thrombosis Mechanisms and Coagulation, Bradykinin, Polyphosphates, and Angioedema.

OpenAlex
World rank
#1,008,916
of 7,710,434 ranked researchers
Rank in United Kingdom
#81,793
of 367,258
Works
134
Citations
5.8k
Citations per work
43.5
h-index
40
i10-index 70Top 4.5% in Cancer Research
Vincent Ellis

What does Vincent Ellis research?

Vincent Ellis's work is mostly in Biochemistry, Genetics and Molecular Biology (35%), Medicine (31%).

Life Sciences: 47 works · 34.9% of their workBiochemistry, Genetics and Molecular Biology: 47 works · 34.9% of their workCancer Research: 29 works · 21.3% of their workMolecular Biology: 12 works · 8.9% of their workCell Biology: 6 works · 4.7% of their workEverything else: 45 works · 33.8% of their workHealth Sciences: 42 works · 31.3% of their workMedicine: 42 works · 31.3% of their workHematology: 27 works · 20.5% of their workOncology: 7 works · 5.5% of their workGenetics: 7 works · 5.3% of their workTHEIR WORKVincent Ellis134works
Life Sciences35%Health Sciences31%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology4734.9%
  2. Everything else4533.8%
    1. Medicine4231.3%
  1. 1Cancer Research21% of their works
  2. 2Hematology20% of their works
  3. 3Molecular Biology9% of their works
  4. 4Oncology6% of their works
  5. 5Genetics5% of their works
  6. 6Cell Biology5% of their works

Research topics

Which keywords describe Vincent Ellis's research?

The keywords of their largest research topics: Angiogenesis, Extracellular Matrix, Inflammation, Cell Signaling, Hemostasis, Matrix Metalloproteinases, Tissue Inhibitors of Metalloproteinases and Protease-Activated Receptors.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 21 words, with their numbers
  1. Angiogenesis1224 topics
  2. Extracellular Matrix942 topics
  3. Inflammation933 topics
  4. Cell Signaling872 topics
  5. Hemostasis772 topics
  6. Matrix Metalloproteinases771 topic
  7. Tissue Inhibitors of Metalloproteinases771 topic
  8. Protease-Activated Receptors661 topic
  9. Thrombin661 topic
  10. Fibroblast Activation Protein181 topic
  11. Hereditary Angioedema181 topic
  12. Kinin Receptor Family181 topic
  13. Tumor Microenvironment181 topic
  14. Cell Adhesion171 topic
  15. Integrins171 topic
  16. Biotechnological Applications121 topic
  17. Microbial Enzymes121 topic
  18. Fibrinogen111 topic
  19. Transglutaminases111 topic
  20. Hyaluronan101 topic
  21. Proteoglycans101 topic

Where does Vincent Ellis work?

Vincent Ellis's main affiliation in the publication record is Thrombosis Research Institute, United Kingdom.

How many publications and citations does Vincent Ellis have?

OpenAlex counts 134 works and 5,831 citations for Vincent Ellis, an h-index of 40, ranking #1,008,916 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

Ask a question or share a note about Vincent Ellis. Keep it to the research: no personal attacks, advertising or links. Any reader can report a post, which collapses it for everyone until it is opened.