Science Explorer Back to mapMap
Discussion
Researcher · Augusta University · United States

Wen‐Cheng Xiong

Wen‐Cheng Xiong publishes mostly in Molecular Biology, Cellular and Molecular Neuroscience and Cell Biology, on topics such as Axon Guidance and Neuronal Signaling, Neuroscience and Neuropharmacology Research and Muscle Physiology and Disorders.

ORCID 0000-0001-9071-7598 OpenAlex
World rank
#106,550
of 7,710,434 ranked researchers
Rank in United States
#40,716
of 1,627,098
Works
301
Citations
22k
Citations per work
72.0
h-index
81
i10-index 212Top 0.6% in Molecular Biology
Wen‐Cheng Xiong

What does Wen‐Cheng Xiong research?

Wen‐Cheng Xiong's work is mostly in Biochemistry, Genetics and Molecular Biology (40%), Neuroscience (18%), Medicine (7%).

Life Sciences: 175 works · 58.1% of their workBiochemistry, Genetics and Molecular Biology: 122 works · 40.4% of their workMolecular Biology: 93 works · 31.0% of their workCell Biology: 28 works · 9.4% of their workNeuroscience: 53 works · 17.7% of their workCellular and Molecular Neuroscience: 43 works · 14.4% of their workDevelopmental Neuroscience: 10 works · 3.3% of their workEverything else: 105 works · 35.0% of their workHealth Sciences: 21 works · 6.9% of their workMedicine: 21 works · 6.9% of their workOncology: 11 works · 3.6% of their workPhysiology: 10 works · 3.3% of their workTHEIR WORKWen‐Cheng Xiong301works
Life Sciences58%Health Sciences7%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology12240.4%
    2. Neuroscience5317.7%
  2. Everything else10535.0%
    1. Medicine216.9%
  1. 1Molecular Biology31% of their works
  2. 2Cellular and Molecular Neuroscience14% of their works
  3. 3Cell Biology9% of their works
  4. 4Oncology4% of their works
  5. 5Developmental Neuroscience3% of their works
  6. 6Physiology3% of their works

Research topics

Which keywords describe Wen‐Cheng Xiong's research?

The keywords of their largest research topics: Cell Signaling, Stem Cells, Eph receptors, Semaphorins, Neurotransmission, Synaptic Plasticity, Mechanotransduction and Muscle Regeneration.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 19 words, with their numbers
  1. Cell Signaling702 topics
  2. Stem Cells693 topics
  3. Eph receptors411 topic
  4. Semaphorins411 topic
  5. Neurotransmission401 topic
  6. Synaptic Plasticity401 topic
  7. Mechanotransduction392 topics
  8. Muscle Regeneration301 topic
  9. Skeletal Muscle Atrophy301 topic
  10. Ion Channels291 topic
  11. Voltage-Gated Channels291 topic
  12. Adult Stem Cells271 topic
  13. Neurogenesis271 topic
  14. Endocytosis241 topic
  15. Membrane Fusion241 topic
  16. Hippo Pathway201 topic
  17. YAP/TAZ201 topic
  18. Cell Mechanics191 topic
  19. Extracellular Matrix191 topic

Where does Wen‐Cheng Xiong work?

Wen‐Cheng Xiong's main affiliation in the publication record is Augusta University, United States.

How many publications and citations does Wen‐Cheng Xiong have?

OpenAlex counts 301 works and 21,681 citations for Wen‐Cheng Xiong, an h-index of 81, ranking #106,550 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

Discussion

Ask a question or share a note about Wen‐Cheng Xiong. Keep it to the research: no personal attacks, advertising or links. Any reader can report a post, which collapses it for everyone until it is opened.