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Researcher · Doshisha Women's College of Liberal Arts · Japan

Yuki Takada‐Takatori

Yuki Takada‐Takatori publishes mostly in Molecular Biology, Cellular and Molecular Neuroscience and Pharmacology, on topics such as Neuroscience and Neuropharmacology Research, Nicotinic Acetylcholine Receptors Study and Genomics, phytochemicals, and oxidative stress.

OpenAlex
World rank
#4,406,168
of 7,710,434 ranked researchers
Rank in Japan
#125,978
of 383,136
Works
50
Citations
1.6k
Citations per work
31.9
h-index
23
i10-index 35Top 26% in Molecular Biology
Yuki Takada‐Takatori

What does Yuki Takada‐Takatori research?

Yuki Takada‐Takatori's work is mostly in Biochemistry, Genetics and Molecular Biology (30%), Neuroscience (22%), Medicine (16%).

Life Sciences: 26 works · 52.4% of their workBiochemistry, Genetics and Molecular Biology: 15 works · 30.3% of their workMolecular Biology: 15 works · 30.3% of their workNeuroscience: 11 works · 22.1% of their workCellular and Molecular Neuroscience: 8 works · 15.2% of their workNeurology: 3 works · 6.9% of their workEverything else: 16 works · 31.8% of their workHealth Sciences: 8 works · 15.8% of their workMedicine: 8 works · 15.8% of their workPharmacology: 4 works · 7.6% of their workPhysiology: 2 works · 4.8% of their workBiochemistry: 2 works · 3.4% of their workTHEIR WORKYukiTakada‐Takatori50works
Life Sciences52%Health Sciences16%

Their six largest subfields, inside their fields and domains; the grey slice is everything else they publish. The interactive view splits every one of their works down to single topics.

The fields and subfields, with their numbers
    1. Biochemistry, Genetics and Molecular Biology1530.3%
    2. Neuroscience1122.1%
  2. Everything else1631.8%
    1. Medicine815.8%
  1. 1Molecular Biology30% of their works
  2. 2Cellular and Molecular Neuroscience15% of their works
  3. 3Pharmacology8% of their works
  4. 4Neurology7% of their works
  5. 5Physiology5% of their works
  6. 6Biochemistry3% of their works

Research topics

Which keywords describe Yuki Takada‐Takatori's research?

The keywords of their largest research topics: Alzheimer's Disease, Crystal Structure, Pharmacology, Neurotransmission, Synaptic Plasticity, Oxidative Stress, Neuronal Signaling and Nicotinic Acetylcholine Receptors.

Size is their works in the topics tagged with each word, from their 8 largest topics. Each links to the topic it comes from most. The words come from the keyword list OpenAlex gives each topic, not from the text of the papers, so a word can appear that none of the papers uses.

All 22 words, with their numbers
  1. Alzheimer's Disease242 topics
  2. Crystal Structure202 topics
  3. Pharmacology182 topics
  4. Neurotransmission171 topic
  5. Synaptic Plasticity171 topic
  6. Oxidative Stress162 topics
  7. Neuronal Signaling131 topic
  8. Nicotinic Acetylcholine Receptors131 topic
  9. Keap1-Nrf2-ARE Pathway121 topic
  10. Nrf2121 topic
  11. Acetylcholinesterase111 topic
  12. Cholinesterase Inhibitors111 topic
  13. Brain Development92 topics
  14. Neuroinflammation92 topics
  15. G-Protein-Coupled Receptors71 topic
  16. Signal Transduction71 topic
  17. Astrocytes51 topic
  18. Microglia51 topic
  19. Adult Stem Cells41 topic
  20. Amyloid Hypothesis41 topic
  21. Neurogenesis41 topic
  22. Tau Pathology41 topic

Where does Yuki Takada‐Takatori work?

Yuki Takada‐Takatori's main affiliation in the publication record is Doshisha Women's College of Liberal Arts, Japan.

How many publications and citations does Yuki Takada‐Takatori have?

OpenAlex counts 50 works and 1,593 citations for Yuki Takada‐Takatori, an h-index of 23, ranking #4,406,168 of 7,710,434 researchers worldwide on the quality score.

Ranked on field-normalised citation excellence (50%), output (30%) and citations (20%). Counts come from OpenAlex author records, which occasionally merge different people who share a name or split one person into several.

Papers, co-authors, who cited this work and researchers on the nearest topics have tabs of their own, above. Is this your page? Claim it with your ORCID iD, or request a correction or removal.

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